Overview
SCIENTIFIC SCORE
Possibly Effective
Based on 7 Researches
7.8
USERS' SCORE
Good
Based on 5 Reviews
8.3
Supplement Facts
Serving Size: 1 Softgel
Amount Per Serving
%DV
Vitamin D3 (as Cholecalciferol) (from Lanolin)
125 mcg (5,000 IU)
625%
Top Medical Research Studies
8
Vitamin D3 benefits gastritis treatment
We investigated the impact of vitamin D3 (VD3) on gastritis, especially concerning Helicobacter pylori (Hp) infections and high cholesterol levels. Mice were used to establish conditions of hypercholesterolemia and Hp infection, allowing us to assess the effectiveness of VD3 over an eight-week treatment period.
Throughout the study, we observed significant improvements in the liver and gastric tissues of mice that received VD3. Specifically, levels of vitamin D receptors and Insig-2 increased in the liver, while serum gastrin levels decreased, indicating a positive response to the treatment. We also noted a reduction in inflammatory markers associated with the JAK/STAT3 signaling pathway, which suggests that VD3 plays a role in alleviating inflammation linked to gastritis.
Importantly, alongside these findings, we observed improvements in liver health, as the treatment reduced hepatic ballooning and gastric tissue inflammation in the high cholesterol and Hp infected groups. Overall, our results illustrate that VD3 is a promising aid in alleviating gastritis related to both high cholesterol and Hp, potentially through its mechanism of enhancing liver health and disrupting inflammatory pathways.
Throughout the study, we observed significant improvements in the liver and gastric tissues of mice that received VD3. Specifically, levels of vitamin D receptors and Insig-2 increased in the liver, while serum gastrin levels decreased, indicating a positive response to the treatment. We also noted a reduction in inflammatory markers associated with the JAK/STAT3 signaling pathway, which suggests that VD3 plays a role in alleviating inflammation linked to gastritis.
Importantly, alongside these findings, we observed improvements in liver health, as the treatment reduced hepatic ballooning and gastric tissue inflammation in the high cholesterol and Hp infected groups. Overall, our results illustrate that VD3 is a promising aid in alleviating gastritis related to both high cholesterol and Hp, potentially through its mechanism of enhancing liver health and disrupting inflammatory pathways.
Read More
9.5
We observed an intriguing case involving an 18-month-old girl who struggled with vomiting, diarrhea, and failure to thrive. Initially, she was exclusively breastfed, and her blood tests revealed a vitamin D deficiency paired with low calcium and phosphorus levels. After regular vitamin D and calcium supplementation was initiated, her condition presented further challenges, including lower-limb bowing and irritability.
As time went on, despite an increase in her vitamin D dosage, her growth velocity stagnated, and gastrointestinal symptoms persisted. To deepen our understanding, an endoscopic examination was performed which identified Helicobacter pylori gastritis, a bacterial infection in the stomach lining. Following the eradication of H. pylori, we noted a significant improvement in her growth and overall activity levels.
Interestingly, after six months of follow-up, there were no signs of rickets, showcasing an impressive turnaround. While we recognize that vitamin D was part of her treatment regimen, it’s essential to note that the significant improvement can largely be attributed to addressing the H. pylori infection. This highlights the complex interplay between vitamin deficiencies and gastrointestinal health in children.
As time went on, despite an increase in her vitamin D dosage, her growth velocity stagnated, and gastrointestinal symptoms persisted. To deepen our understanding, an endoscopic examination was performed which identified Helicobacter pylori gastritis, a bacterial infection in the stomach lining. Following the eradication of H. pylori, we noted a significant improvement in her growth and overall activity levels.
Interestingly, after six months of follow-up, there were no signs of rickets, showcasing an impressive turnaround. While we recognize that vitamin D was part of her treatment regimen, it’s essential to note that the significant improvement can largely be attributed to addressing the H. pylori infection. This highlights the complex interplay between vitamin deficiencies and gastrointestinal health in children.
Read More
8
We explored the effect of vitamin D levels on the success of treatment for gastritis, particularly focusing on the bacteria known as H. pylori. In our study, we included 150 patients diagnosed with gastritis, measuring their serum 25-hydroxy-vitamin D (25[OH]D) levels before starting a 14-day clarithromycin-based triple therapy.
After treatment, we assessed whether the bacteria were successfully eradicated using a stool antigen test. Our findings showed that a significant number of patients, specifically 70%, successfully eradicated the infection, while 30% did not.
Notably, we observed that those who had low vitamin D levels—defined as less than 20 ng/mL—had higher rates of treatment failure compared to those with sufficient vitamin D levels. This suggests that vitamin D deficiency could be a factor affecting the success of gastritis treatment.
Overall, our results highlight the potential importance of vitamin D in improving treatment outcomes for gastritis linked to H. pylori. We believe further research, particularly randomized trials assessing vitamin D supplementation in this context, is necessary to better understand its role.
After treatment, we assessed whether the bacteria were successfully eradicated using a stool antigen test. Our findings showed that a significant number of patients, specifically 70%, successfully eradicated the infection, while 30% did not.
Notably, we observed that those who had low vitamin D levels—defined as less than 20 ng/mL—had higher rates of treatment failure compared to those with sufficient vitamin D levels. This suggests that vitamin D deficiency could be a factor affecting the success of gastritis treatment.
Overall, our results highlight the potential importance of vitamin D in improving treatment outcomes for gastritis linked to H. pylori. We believe further research, particularly randomized trials assessing vitamin D supplementation in this context, is necessary to better understand its role.
Read More
Most Useful Reviews
9
Successful vitamin D boost
Excellent product! After battling a severe vitamin D deficiency, I began with 5,000 for a month, then 10,000 for two months. My vitamin D levels rose significantly, and I experienced no pain or heartburn, even with gastritis.
Read More
7.5
Mood improvement
1 people found this helpful
A good product; my husband and I have already consumed several packages. My mood is excellent, and my vitamin levels have increased, with no side effects despite my gastritis.
Read More
7.5
Stomach-friendly capsules
The capsules are a good size and do not negatively affect my chronic gastritis. They are easy to swallow, and I can adjust my daily dosage as needed.
Read More
Medical Researches
SCIENTIFIC SCORE
Possibly Effective
Based on 7 Researches
7.8
- All Researches
9.5
We observed an intriguing case involving an 18-month-old girl who struggled with vomiting, diarrhea, and failure to thrive. Initially, she was exclusively breastfed, and her blood tests revealed a vitamin D deficiency paired with low calcium and phosphorus levels. After regular vitamin D and calcium supplementation was initiated, her condition presented further challenges, including lower-limb bowing and irritability.
As time went on, despite an increase in her vitamin D dosage, her growth velocity stagnated, and gastrointestinal symptoms persisted. To deepen our understanding, an endoscopic examination was performed which identified Helicobacter pylori gastritis, a bacterial infection in the stomach lining. Following the eradication of H. pylori, we noted a significant improvement in her growth and overall activity levels.
Interestingly, after six months of follow-up, there were no signs of rickets, showcasing an impressive turnaround. While we recognize that vitamin D was part of her treatment regimen, it’s essential to note that the significant improvement can largely be attributed to addressing the H. pylori infection. This highlights the complex interplay between vitamin deficiencies and gastrointestinal health in children.
As time went on, despite an increase in her vitamin D dosage, her growth velocity stagnated, and gastrointestinal symptoms persisted. To deepen our understanding, an endoscopic examination was performed which identified Helicobacter pylori gastritis, a bacterial infection in the stomach lining. Following the eradication of H. pylori, we noted a significant improvement in her growth and overall activity levels.
Interestingly, after six months of follow-up, there were no signs of rickets, showcasing an impressive turnaround. While we recognize that vitamin D was part of her treatment regimen, it’s essential to note that the significant improvement can largely be attributed to addressing the H. pylori infection. This highlights the complex interplay between vitamin deficiencies and gastrointestinal health in children.
Read More
8
Vitamin D3 benefits gastritis treatment
We investigated the impact of vitamin D3 (VD3) on gastritis, especially concerning Helicobacter pylori (Hp) infections and high cholesterol levels. Mice were used to establish conditions of hypercholesterolemia and Hp infection, allowing us to assess the effectiveness of VD3 over an eight-week treatment period.
Throughout the study, we observed significant improvements in the liver and gastric tissues of mice that received VD3. Specifically, levels of vitamin D receptors and Insig-2 increased in the liver, while serum gastrin levels decreased, indicating a positive response to the treatment. We also noted a reduction in inflammatory markers associated with the JAK/STAT3 signaling pathway, which suggests that VD3 plays a role in alleviating inflammation linked to gastritis.
Importantly, alongside these findings, we observed improvements in liver health, as the treatment reduced hepatic ballooning and gastric tissue inflammation in the high cholesterol and Hp infected groups. Overall, our results illustrate that VD3 is a promising aid in alleviating gastritis related to both high cholesterol and Hp, potentially through its mechanism of enhancing liver health and disrupting inflammatory pathways.
Throughout the study, we observed significant improvements in the liver and gastric tissues of mice that received VD3. Specifically, levels of vitamin D receptors and Insig-2 increased in the liver, while serum gastrin levels decreased, indicating a positive response to the treatment. We also noted a reduction in inflammatory markers associated with the JAK/STAT3 signaling pathway, which suggests that VD3 plays a role in alleviating inflammation linked to gastritis.
Importantly, alongside these findings, we observed improvements in liver health, as the treatment reduced hepatic ballooning and gastric tissue inflammation in the high cholesterol and Hp infected groups. Overall, our results illustrate that VD3 is a promising aid in alleviating gastritis related to both high cholesterol and Hp, potentially through its mechanism of enhancing liver health and disrupting inflammatory pathways.
Read More
8
We explored whether adding vitamin D3 to standard treatment for Helicobacter pylori (H. pylori) could help improve eradication rates in patients with gastritis. In a recent study, 150 patients diagnosed with H. pylori gastritis were divided into two groups. One group received the standard clarithromycin-based treatment, while the other group was given vitamin D3 along with the same antibiotics.
After the treatment, we assessed the success of eradication using stool tests conducted four weeks later. Results indicated that adding vitamin D3 led to significantly higher eradication rates of H. pylori compared to the standard treatment alone. This suggests that vitamin D3 may play an additional role in enhancing treatment effectiveness.
However, it's important to note that by integrating vitamin D3 into a multi-faceted treatment plan, we cannot fully isolate its independent effects on gastritis. Still, the findings highlight the potential benefits of incorporating vitamin D3 in conventional therapy for better outcomes.
After the treatment, we assessed the success of eradication using stool tests conducted four weeks later. Results indicated that adding vitamin D3 led to significantly higher eradication rates of H. pylori compared to the standard treatment alone. This suggests that vitamin D3 may play an additional role in enhancing treatment effectiveness.
However, it's important to note that by integrating vitamin D3 into a multi-faceted treatment plan, we cannot fully isolate its independent effects on gastritis. Still, the findings highlight the potential benefits of incorporating vitamin D3 in conventional therapy for better outcomes.
Read More
8
We explored the effect of vitamin D levels on the success of treatment for gastritis, particularly focusing on the bacteria known as H. pylori. In our study, we included 150 patients diagnosed with gastritis, measuring their serum 25-hydroxy-vitamin D (25[OH]D) levels before starting a 14-day clarithromycin-based triple therapy.
After treatment, we assessed whether the bacteria were successfully eradicated using a stool antigen test. Our findings showed that a significant number of patients, specifically 70%, successfully eradicated the infection, while 30% did not.
Notably, we observed that those who had low vitamin D levels—defined as less than 20 ng/mL—had higher rates of treatment failure compared to those with sufficient vitamin D levels. This suggests that vitamin D deficiency could be a factor affecting the success of gastritis treatment.
Overall, our results highlight the potential importance of vitamin D in improving treatment outcomes for gastritis linked to H. pylori. We believe further research, particularly randomized trials assessing vitamin D supplementation in this context, is necessary to better understand its role.
After treatment, we assessed whether the bacteria were successfully eradicated using a stool antigen test. Our findings showed that a significant number of patients, specifically 70%, successfully eradicated the infection, while 30% did not.
Notably, we observed that those who had low vitamin D levels—defined as less than 20 ng/mL—had higher rates of treatment failure compared to those with sufficient vitamin D levels. This suggests that vitamin D deficiency could be a factor affecting the success of gastritis treatment.
Overall, our results highlight the potential importance of vitamin D in improving treatment outcomes for gastritis linked to H. pylori. We believe further research, particularly randomized trials assessing vitamin D supplementation in this context, is necessary to better understand its role.
Read More
7
We examined the connection between vitamin D deficiency and chronic autoimmune atrophic gastritis (CAAG), a condition that hampers vitamin absorption due to changes in stomach function. In our study, we evaluated 87 patients diagnosed with CAAG, focusing on their vitamin D levels compared to a control group of 1,232 healthy individuals.
Our findings revealed a significant reduction in vitamin D, specifically the 25-OH-Vitamin D levels, among the CAAG patients. The average 25-OH-Vitamin D level was 18.8 ng/ml in CAAG patients, markedly lower than the 27.0 ng/ml found in healthy individuals. Moreover, we discovered that more than half of the patients had levels below the normal threshold, which might indicate an issue with vitamin absorption due to their condition.
Interestingly, this study also highlighted a possible correlation between lower vitamin D levels and the severity of gastric atrophy, as patients with moderate or severe atrophy had even lower vitamin D levels than those with milder forms. Additionally, patients with gastric carcinoids exhibited the lowest vitamin D levels, suggesting a potential relationship between vitamin D deficiency and certain complications of CAAG.
Despite our insights into vitamin D deficiency in CAAG, we didn't delve deeply into the effects of vitamin D treatment on gastritis specifically. Therefore, while there seems to be a notable deficit in vitamin D among these patients, the implications for treatment remain unclear, emphasizing an area for future exploration.
Our findings revealed a significant reduction in vitamin D, specifically the 25-OH-Vitamin D levels, among the CAAG patients. The average 25-OH-Vitamin D level was 18.8 ng/ml in CAAG patients, markedly lower than the 27.0 ng/ml found in healthy individuals. Moreover, we discovered that more than half of the patients had levels below the normal threshold, which might indicate an issue with vitamin absorption due to their condition.
Interestingly, this study also highlighted a possible correlation between lower vitamin D levels and the severity of gastric atrophy, as patients with moderate or severe atrophy had even lower vitamin D levels than those with milder forms. Additionally, patients with gastric carcinoids exhibited the lowest vitamin D levels, suggesting a potential relationship between vitamin D deficiency and certain complications of CAAG.
Despite our insights into vitamin D deficiency in CAAG, we didn't delve deeply into the effects of vitamin D treatment on gastritis specifically. Therefore, while there seems to be a notable deficit in vitamin D among these patients, the implications for treatment remain unclear, emphasizing an area for future exploration.
Read More
User Reviews
USERS' SCORE
Good
Based on 5 Reviews
8.3
- All Reviews
- Positive Reviews
- Negative Reviews
9
Successful vitamin D boost
Excellent product! After battling a severe vitamin D deficiency, I began with 5,000 for a month, then 10,000 for two months. My vitamin D levels rose significantly, and I experienced no pain or heartburn, even with gastritis.
Read More
7.5
Mood improvement
1 people found this helpful
A good product; my husband and I have already consumed several packages. My mood is excellent, and my vitamin levels have increased, with no side effects despite my gastritis.
Read More
7.5
Stomach-friendly capsules
The capsules are a good size and do not negatively affect my chronic gastritis. They are easy to swallow, and I can adjust my daily dosage as needed.
Read More
7.5
No adverse reactions
Perfect! This vitamin works well with my gastritis. The small capsules are easy to swallow and cause no unpleasant aftertaste. My test results show they are effective, but remember that excess vitamin D can be dangerous.
Read More
7.5
Easily absorbed vitamins
Small capsules are easy to take, as advised by my gastroenterologist. They enhance absorption, especially beneficial for my gastritis. A jar lasts long, and I take 10,000 daily with omega-3 during breakfast.
Read More
Frequently Asked Questions
9
Successful vitamin D boost
Excellent product! After battling a severe vitamin D deficiency, I began with 5,000 for a month, then 10,000 for two months. My vitamin D levels rose significantly, and I experienced no pain or heartburn, even with gastritis.
7.5
Mood improvement
1 people found this helpful
A good product; my husband and I have already consumed several packages. My mood is excellent, and my vitamin levels have increased, with no side effects despite my gastritis.
7.5
Stomach-friendly capsules
The capsules are a good size and do not negatively affect my chronic gastritis. They are easy to swallow, and I can adjust my daily dosage as needed.
7.5
Easily absorbed vitamins
Small capsules are easy to take, as advised by my gastroenterologist. They enhance absorption, especially beneficial for my gastritis. A jar lasts long, and I take 10,000 daily with omega-3 during breakfast.
7.5
No adverse reactions
Perfect! This vitamin works well with my gastritis. The small capsules are easy to swallow and cause no unpleasant aftertaste. My test results show they are effective, but remember that excess vitamin D can be dangerous.
8
We explored whether adding vitamin D3 to standard treatment for Helicobacter pylori (H. pylori) could help improve eradication rates in patients with gastritis. In a recent study, 150 patients diagnosed with H. pylori gastritis were divided into two groups. One group received the standard clarithromycin-based treatment, while the other group was given vitamin D3 along with the same antibiotics.
After the treatment, we assessed the success of eradication using stool tests conducted four weeks later. Results indicated that adding vitamin D3 led to significantly higher eradication rates of H. pylori compared to the standard treatment alone. This suggests that vitamin D3 may play an additional role in enhancing treatment effectiveness.
However, it's important to note that by integrating vitamin D3 into a multi-faceted treatment plan, we cannot fully isolate its independent effects on gastritis. Still, the findings highlight the potential benefits of incorporating vitamin D3 in conventional therapy for better outcomes.
After the treatment, we assessed the success of eradication using stool tests conducted four weeks later. Results indicated that adding vitamin D3 led to significantly higher eradication rates of H. pylori compared to the standard treatment alone. This suggests that vitamin D3 may play an additional role in enhancing treatment effectiveness.
However, it's important to note that by integrating vitamin D3 into a multi-faceted treatment plan, we cannot fully isolate its independent effects on gastritis. Still, the findings highlight the potential benefits of incorporating vitamin D3 in conventional therapy for better outcomes.
7
We explored how Vitamin D might interact with gastritis, especially when caused by the gastric pathogen Helicobacter pylori. In a detailed study involving 17 patients infected with H. pylori and 16 control participants, we looked at the expression of the vitamin D receptor (VDR) within the gastric mucosa.
Our focus was on understanding if increased levels of VDR could play a role in combating gastritis linked to H. pylori infection. We gathered data by measuring various immune response markers in both infected and healthy gastric tissues. Notably, we discovered that VDR expression and the cathelicidin antimicrobial protein (CAMP) were significantly elevated in those with H. pylori infection, revealing a potential defensive mechanism at play.
Additionally, treatment with 1α,25(OH)2 D3, an active form of vitamin D, boosted CAMP expression while reducing certain inflammatory cytokines in gastric cells. This suggests that vitamin D may enhance the body's ability to fend off the bacteria. However, it's important to note that while VDR has been shown to support the immune response, more research is needed to fully understand its therapeutic potential.
In summary, our findings indicate that vitamin D could play a significant role in managing inflammation and supporting gastric health in those affected by H. pylori, offering promising insight into possible preventative strategies.
Our focus was on understanding if increased levels of VDR could play a role in combating gastritis linked to H. pylori infection. We gathered data by measuring various immune response markers in both infected and healthy gastric tissues. Notably, we discovered that VDR expression and the cathelicidin antimicrobial protein (CAMP) were significantly elevated in those with H. pylori infection, revealing a potential defensive mechanism at play.
Additionally, treatment with 1α,25(OH)2 D3, an active form of vitamin D, boosted CAMP expression while reducing certain inflammatory cytokines in gastric cells. This suggests that vitamin D may enhance the body's ability to fend off the bacteria. However, it's important to note that while VDR has been shown to support the immune response, more research is needed to fully understand its therapeutic potential.
In summary, our findings indicate that vitamin D could play a significant role in managing inflammation and supporting gastric health in those affected by H. pylori, offering promising insight into possible preventative strategies.
8
We explored the effect of vitamin D levels on the success of treatment for gastritis, particularly focusing on the bacteria known as H. pylori. In our study, we included 150 patients diagnosed with gastritis, measuring their serum 25-hydroxy-vitamin D (25[OH]D) levels before starting a 14-day clarithromycin-based triple therapy.
After treatment, we assessed whether the bacteria were successfully eradicated using a stool antigen test. Our findings showed that a significant number of patients, specifically 70%, successfully eradicated the infection, while 30% did not.
Notably, we observed that those who had low vitamin D levels—defined as less than 20 ng/mL—had higher rates of treatment failure compared to those with sufficient vitamin D levels. This suggests that vitamin D deficiency could be a factor affecting the success of gastritis treatment.
Overall, our results highlight the potential importance of vitamin D in improving treatment outcomes for gastritis linked to H. pylori. We believe further research, particularly randomized trials assessing vitamin D supplementation in this context, is necessary to better understand its role.
After treatment, we assessed whether the bacteria were successfully eradicated using a stool antigen test. Our findings showed that a significant number of patients, specifically 70%, successfully eradicated the infection, while 30% did not.
Notably, we observed that those who had low vitamin D levels—defined as less than 20 ng/mL—had higher rates of treatment failure compared to those with sufficient vitamin D levels. This suggests that vitamin D deficiency could be a factor affecting the success of gastritis treatment.
Overall, our results highlight the potential importance of vitamin D in improving treatment outcomes for gastritis linked to H. pylori. We believe further research, particularly randomized trials assessing vitamin D supplementation in this context, is necessary to better understand its role.
8
Vitamin D3 benefits gastritis treatment
We investigated the impact of vitamin D3 (VD3) on gastritis, especially concerning Helicobacter pylori (Hp) infections and high cholesterol levels. Mice were used to establish conditions of hypercholesterolemia and Hp infection, allowing us to assess the effectiveness of VD3 over an eight-week treatment period.
Throughout the study, we observed significant improvements in the liver and gastric tissues of mice that received VD3. Specifically, levels of vitamin D receptors and Insig-2 increased in the liver, while serum gastrin levels decreased, indicating a positive response to the treatment. We also noted a reduction in inflammatory markers associated with the JAK/STAT3 signaling pathway, which suggests that VD3 plays a role in alleviating inflammation linked to gastritis.
Importantly, alongside these findings, we observed improvements in liver health, as the treatment reduced hepatic ballooning and gastric tissue inflammation in the high cholesterol and Hp infected groups. Overall, our results illustrate that VD3 is a promising aid in alleviating gastritis related to both high cholesterol and Hp, potentially through its mechanism of enhancing liver health and disrupting inflammatory pathways.
Throughout the study, we observed significant improvements in the liver and gastric tissues of mice that received VD3. Specifically, levels of vitamin D receptors and Insig-2 increased in the liver, while serum gastrin levels decreased, indicating a positive response to the treatment. We also noted a reduction in inflammatory markers associated with the JAK/STAT3 signaling pathway, which suggests that VD3 plays a role in alleviating inflammation linked to gastritis.
Importantly, alongside these findings, we observed improvements in liver health, as the treatment reduced hepatic ballooning and gastric tissue inflammation in the high cholesterol and Hp infected groups. Overall, our results illustrate that VD3 is a promising aid in alleviating gastritis related to both high cholesterol and Hp, potentially through its mechanism of enhancing liver health and disrupting inflammatory pathways.
References
- Xu W, Wang L, Yang L, Li X, Li C, et al. [Vitamin D3 alleviates the gastritis that associated with Helicobacter pylori infection in mice with hypercholesterolemia by enhancing the activity of vitamin D receptors in the liver tissue and blocking the signaling pathway of JAK/STAT3]. Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi. 2024;40:520.
- El Shahawy MS, Shady ZM, Gaafar A. Influence of adding vitamin D3 to standard clarithromycin-based triple therapy on the eradication rates of Helicobacter pylori infection. Arab J Gastroenterol. 2021;22:209. doi:10.1016/j.ajg.2021.08.002
- El Shahawy MS, Hemida MH, El Metwaly I, Shady ZM. The effect of vitamin D deficiency on eradication rates of infection. JGH Open. 2018;2:270. doi:10.1002/jgh3.12081
- Massironi S, Cavalcoli F, Zilli A, Del Gobbo A, Ciafardini C, et al. Relevance of vitamin D deficiency in patients with chronic autoimmune atrophic gastritis: a prospective study. BMC Gastroenterol. 2018;18:172. doi:10.1186/s12876-018-0901-0
- Yildirim O, Yildirim T, Seckin Y, Osanmaz P, Bilgic Y, et al. The influence of vitamin D deficiency on eradication rates of Helicobacter pylori. Adv Clin Exp Med. 2017;26:1377. doi:10.17219/acem/65430
- Guo L, Chen W, Zhu H, Chen Y, Wan X, et al. Helicobacter pylori induces increased expression of the vitamin d receptor in immune responses. Helicobacter. 2014;19:37. doi:10.1111/hel.12102
- Bharwani SS, Shaukat Q, Balhaj G, Ashari M. A failing to thrive 18 month old with vitamin D deficiency rickets and Helicobacter pylori gastritis. BMJ Case Rep. 2011;2011. doi:10.1136/bcr.04.2011.4160
