Last update
4/9/2026
Reviewed By
Overview
SCIENTIFIC SCORE
Questionable
Based on 29 Researches
6.7
USERS' SCORE
Good
Based on 3 Reviews
8.2
Supplement Facts
Serving Size: 3 Tablets
Amount Per Serving
%DV
Vitamin B6 (as pyridoxine HCl)
25 mg
1,471%
Magnesium (as magnesium oxide)
400 mg
95%
Top Medical Research Studies
9
We explored the potential of magnesium implantation as a new approach to combat heart attacks, specifically myocardial infarction (MI). Our research centered on using magnesium slices implanted under the skin of rats to generate hydrogen gas continuously. This method could ensure a greater and longer-lasting concentration of hydrogen directly reaching the heart, which is crucial for its therapeutic effects.
Through our study, we measured how effectively the magnesium produced hydrogen and evaluated its safety. The results were promising; we observed that magnesium implantation not only improved cardiac function in rats suffering from MI, but also played a role in eliminating harmful free radicals from mitochondrial dysfunction. Furthermore, it helped reduce cell death in heart muscle cells, which is a common consequence of heart attacks.
Compared to traditional hydrogen inhalation, which has limitations in delivering sufficient doses over time, magnesium implantation proved to be a superior method. Overall, our findings pave the way for innovative treatments that could enhance recovery after heart attacks by harnessing the unique properties of magnesium for hydrogen production.
Through our study, we measured how effectively the magnesium produced hydrogen and evaluated its safety. The results were promising; we observed that magnesium implantation not only improved cardiac function in rats suffering from MI, but also played a role in eliminating harmful free radicals from mitochondrial dysfunction. Furthermore, it helped reduce cell death in heart muscle cells, which is a common consequence of heart attacks.
Compared to traditional hydrogen inhalation, which has limitations in delivering sufficient doses over time, magnesium implantation proved to be a superior method. Overall, our findings pave the way for innovative treatments that could enhance recovery after heart attacks by harnessing the unique properties of magnesium for hydrogen production.
Read More
9
We explored the potential of vitamin B6 (VB6) in promoting recovery after heart attacks, specifically its role in angiogenesis, which is the formation of new blood vessels. The study aimed to determine whether VB6 could help prevent cardiac dysfunction following acute myocardial infarction (AMI).
To assess the effects of VB6, we conducted both in vitro experiments, examining endothelial cell behavior, and in vivo tests using mice with heart attacks. We discovered that VB6 significantly enhanced cell migration and tubule formation in human umbilical vein endothelial cells, which are critical for blood vessel formation. This process was linked to increased activity of a protein called AMP-activated protein kinase (AMPK).
Interestingly, our findings showed that these beneficial effects of VB6 were reversed when we introduced AMPK inhibitors. This leads us to conclude that VB6 promotes heart recovery by activating AMPK, which in turn supports angiogenesis following AMI. In practical terms, long-term VB6 supplementation after heart attacks led to improved heart function and increased new blood vessel formation in mice, making this vitamin a promising candidate for heart recovery therapies.
To assess the effects of VB6, we conducted both in vitro experiments, examining endothelial cell behavior, and in vivo tests using mice with heart attacks. We discovered that VB6 significantly enhanced cell migration and tubule formation in human umbilical vein endothelial cells, which are critical for blood vessel formation. This process was linked to increased activity of a protein called AMP-activated protein kinase (AMPK).
Interestingly, our findings showed that these beneficial effects of VB6 were reversed when we introduced AMPK inhibitors. This leads us to conclude that VB6 promotes heart recovery by activating AMPK, which in turn supports angiogenesis following AMI. In practical terms, long-term VB6 supplementation after heart attacks led to improved heart function and increased new blood vessel formation in mice, making this vitamin a promising candidate for heart recovery therapies.
Read More
9
We investigated the potential of pyridoxamine, a form of vitamin B6, to improve outcomes after a heart attack (myocardial infarction, or MI). In our study, we divided rats into three groups: one that suffered from MI, another that also received pyridoxamine, and a sham group for comparison.
Over the course of eight weeks, we observed how these treatments impacted heart function using echocardiography and hemodynamic assessments. Remarkably, we found that pyridoxamine not only enhanced survival rates post-heart attack but also significantly reduced harmful levels of advanced glycation end products (AGEs) – compounds that can lead to heart failure.
Specifically, rats treated with pyridoxamine exhibited lower left ventricular pressures and improved heart deformation parameters compared to untreated rats. This better heart function was linked to a decrease in collagen in heart tissue, especially around the damaged area, which is crucial because excess collagen can worsen heart stiffness.
Overall, our findings suggest that pyridoxamine could be a promising therapy for preventing detrimental heart changes following a heart attack, highlighting the value of targeting AGEs in treatment strategies.
Over the course of eight weeks, we observed how these treatments impacted heart function using echocardiography and hemodynamic assessments. Remarkably, we found that pyridoxamine not only enhanced survival rates post-heart attack but also significantly reduced harmful levels of advanced glycation end products (AGEs) – compounds that can lead to heart failure.
Specifically, rats treated with pyridoxamine exhibited lower left ventricular pressures and improved heart deformation parameters compared to untreated rats. This better heart function was linked to a decrease in collagen in heart tissue, especially around the damaged area, which is crucial because excess collagen can worsen heart stiffness.
Overall, our findings suggest that pyridoxamine could be a promising therapy for preventing detrimental heart changes following a heart attack, highlighting the value of targeting AGEs in treatment strategies.
Read More
Most Useful Reviews
7.5
Less painful cramps
I took this magnesium during my pregnancy as I was plagued by night cramps. With this supplement, the frequency and pain of my nighttime attacks decreased significantly.
Read More
7.5
Heart pain relief
After recovering from Covid, I experienced occasional heart pain. Just a few days of taking this supplement alleviated the discomfort entirely!
Read More
6
Surge of strength
I really appreciate Solgar's products due to their high quality. I purchased their magnesium B6, and the characteristics listed were accurate. I've felt a surge of strength, my sleep has improved, and my heart feels calmer now. I highly recommend it!
Read More
Medical Researches
SCIENTIFIC SCORE
Questionable
Based on 29 Researches
6.7
- All Researches
9
We explored the potential of magnesium implantation as a new approach to combat heart attacks, specifically myocardial infarction (MI). Our research centered on using magnesium slices implanted under the skin of rats to generate hydrogen gas continuously. This method could ensure a greater and longer-lasting concentration of hydrogen directly reaching the heart, which is crucial for its therapeutic effects.
Through our study, we measured how effectively the magnesium produced hydrogen and evaluated its safety. The results were promising; we observed that magnesium implantation not only improved cardiac function in rats suffering from MI, but also played a role in eliminating harmful free radicals from mitochondrial dysfunction. Furthermore, it helped reduce cell death in heart muscle cells, which is a common consequence of heart attacks.
Compared to traditional hydrogen inhalation, which has limitations in delivering sufficient doses over time, magnesium implantation proved to be a superior method. Overall, our findings pave the way for innovative treatments that could enhance recovery after heart attacks by harnessing the unique properties of magnesium for hydrogen production.
Through our study, we measured how effectively the magnesium produced hydrogen and evaluated its safety. The results were promising; we observed that magnesium implantation not only improved cardiac function in rats suffering from MI, but also played a role in eliminating harmful free radicals from mitochondrial dysfunction. Furthermore, it helped reduce cell death in heart muscle cells, which is a common consequence of heart attacks.
Compared to traditional hydrogen inhalation, which has limitations in delivering sufficient doses over time, magnesium implantation proved to be a superior method. Overall, our findings pave the way for innovative treatments that could enhance recovery after heart attacks by harnessing the unique properties of magnesium for hydrogen production.
Read More
9
Magnesium scaffolds improve heart attack outcomes
We examined the performance of two types of bioresorbable scaffolds in patients experiencing acute coronary syndrome (ACS) - a common and serious manifestation of heart disease. Specifically, we compared the effectiveness of magnesium-based scaffolds, known as Magmaris, with the earlier polymer scaffolds called Absorb.
Our study included 193 patients using Magmaris and 160 patients using Absorb, all treated in similar clinical settings. Over the course of one year, we monitored their health outcomes, particularly focusing on serious issues like cardiac death, heart attacks, and instances of scaffold thrombosis.
Surprisingly, we found that patients with Magmaris had significantly better outcomes than those with Absorb. Specifically, the rates of primary complications and target lesion failures were notably lower with Magmaris. We noted a concerning trend with Absorb, where patients experienced a higher rate of scaffold thrombosis.
Overall, our findings suggest that magnesium-based scaffolds could provide a safer and more effective option for patients undergoing treatment for heart attacks compared to their polymer counterparts.
Our study included 193 patients using Magmaris and 160 patients using Absorb, all treated in similar clinical settings. Over the course of one year, we monitored their health outcomes, particularly focusing on serious issues like cardiac death, heart attacks, and instances of scaffold thrombosis.
Surprisingly, we found that patients with Magmaris had significantly better outcomes than those with Absorb. Specifically, the rates of primary complications and target lesion failures were notably lower with Magmaris. We noted a concerning trend with Absorb, where patients experienced a higher rate of scaffold thrombosis.
Overall, our findings suggest that magnesium-based scaffolds could provide a safer and more effective option for patients undergoing treatment for heart attacks compared to their polymer counterparts.
Read More
9
We explored the potential of vitamin B6 (VB6) in promoting recovery after heart attacks, specifically its role in angiogenesis, which is the formation of new blood vessels. The study aimed to determine whether VB6 could help prevent cardiac dysfunction following acute myocardial infarction (AMI).
To assess the effects of VB6, we conducted both in vitro experiments, examining endothelial cell behavior, and in vivo tests using mice with heart attacks. We discovered that VB6 significantly enhanced cell migration and tubule formation in human umbilical vein endothelial cells, which are critical for blood vessel formation. This process was linked to increased activity of a protein called AMP-activated protein kinase (AMPK).
Interestingly, our findings showed that these beneficial effects of VB6 were reversed when we introduced AMPK inhibitors. This leads us to conclude that VB6 promotes heart recovery by activating AMPK, which in turn supports angiogenesis following AMI. In practical terms, long-term VB6 supplementation after heart attacks led to improved heart function and increased new blood vessel formation in mice, making this vitamin a promising candidate for heart recovery therapies.
To assess the effects of VB6, we conducted both in vitro experiments, examining endothelial cell behavior, and in vivo tests using mice with heart attacks. We discovered that VB6 significantly enhanced cell migration and tubule formation in human umbilical vein endothelial cells, which are critical for blood vessel formation. This process was linked to increased activity of a protein called AMP-activated protein kinase (AMPK).
Interestingly, our findings showed that these beneficial effects of VB6 were reversed when we introduced AMPK inhibitors. This leads us to conclude that VB6 promotes heart recovery by activating AMPK, which in turn supports angiogenesis following AMI. In practical terms, long-term VB6 supplementation after heart attacks led to improved heart function and increased new blood vessel formation in mice, making this vitamin a promising candidate for heart recovery therapies.
Read More
9
We investigated the potential of pyridoxamine, a form of vitamin B6, to improve outcomes after a heart attack (myocardial infarction, or MI). In our study, we divided rats into three groups: one that suffered from MI, another that also received pyridoxamine, and a sham group for comparison.
Over the course of eight weeks, we observed how these treatments impacted heart function using echocardiography and hemodynamic assessments. Remarkably, we found that pyridoxamine not only enhanced survival rates post-heart attack but also significantly reduced harmful levels of advanced glycation end products (AGEs) – compounds that can lead to heart failure.
Specifically, rats treated with pyridoxamine exhibited lower left ventricular pressures and improved heart deformation parameters compared to untreated rats. This better heart function was linked to a decrease in collagen in heart tissue, especially around the damaged area, which is crucial because excess collagen can worsen heart stiffness.
Overall, our findings suggest that pyridoxamine could be a promising therapy for preventing detrimental heart changes following a heart attack, highlighting the value of targeting AGEs in treatment strategies.
Over the course of eight weeks, we observed how these treatments impacted heart function using echocardiography and hemodynamic assessments. Remarkably, we found that pyridoxamine not only enhanced survival rates post-heart attack but also significantly reduced harmful levels of advanced glycation end products (AGEs) – compounds that can lead to heart failure.
Specifically, rats treated with pyridoxamine exhibited lower left ventricular pressures and improved heart deformation parameters compared to untreated rats. This better heart function was linked to a decrease in collagen in heart tissue, especially around the damaged area, which is crucial because excess collagen can worsen heart stiffness.
Overall, our findings suggest that pyridoxamine could be a promising therapy for preventing detrimental heart changes following a heart attack, highlighting the value of targeting AGEs in treatment strategies.
Read More
8
Magnesium enhances heart attack recovery
We explored an innovative approach to treating myocardial infarction (MI) by using a special composite hydrogel that incorporates magnesium-modified black phosphorus nanosheets. This treatment aims to address the significant changes in the heart's environment after a heart attack, which are usually detrimental to recovery.
The research involved creating a hydrogel scaffold that allows for the gradual release of these magnesium-enhanced nanosheets directly at the site of the infarct. This is particularly important because sustained release helps to manage oxidative stress and inflammation, both common issues following a heart attack.
We observed that the black phosphorus nanosheets have a high reactivity with reactive oxygen species (ROS), which helps reduce oxidative stress and inflammation in the heart. Additionally, the magnesium contributes to promoting blood vessel growth, or angiogenesis, which is crucial for long-term heart function after an MI.
Overall, the findings of this study suggest that the combination of these elements in the hydrogel can effectively block further damage from the heart attack and improve recovery outcomes, signaling a promising avenue for heart attack treatments in the future.
The research involved creating a hydrogel scaffold that allows for the gradual release of these magnesium-enhanced nanosheets directly at the site of the infarct. This is particularly important because sustained release helps to manage oxidative stress and inflammation, both common issues following a heart attack.
We observed that the black phosphorus nanosheets have a high reactivity with reactive oxygen species (ROS), which helps reduce oxidative stress and inflammation in the heart. Additionally, the magnesium contributes to promoting blood vessel growth, or angiogenesis, which is crucial for long-term heart function after an MI.
Overall, the findings of this study suggest that the combination of these elements in the hydrogel can effectively block further damage from the heart attack and improve recovery outcomes, signaling a promising avenue for heart attack treatments in the future.
Read More
User Reviews
USERS' SCORE
Good
Based on 3 Reviews
8.2
- All Reviews
- Positive Reviews
- Negative Reviews
7.5
Less painful cramps
I took this magnesium during my pregnancy as I was plagued by night cramps. With this supplement, the frequency and pain of my nighttime attacks decreased significantly.
Read More
7.5
Heart pain relief
After recovering from Covid, I experienced occasional heart pain. Just a few days of taking this supplement alleviated the discomfort entirely!
Read More
6
Surge of strength
I really appreciate Solgar's products due to their high quality. I purchased their magnesium B6, and the characteristics listed were accurate. I've felt a surge of strength, my sleep has improved, and my heart feels calmer now. I highly recommend it!
Read More
Frequently Asked Questions
7.5
Heart pain relief
After recovering from Covid, I experienced occasional heart pain. Just a few days of taking this supplement alleviated the discomfort entirely!
7.5
Less painful cramps
I took this magnesium during my pregnancy as I was plagued by night cramps. With this supplement, the frequency and pain of my nighttime attacks decreased significantly.
6
Surge of strength
I really appreciate Solgar's products due to their high quality. I purchased their magnesium B6, and the characteristics listed were accurate. I've felt a surge of strength, my sleep has improved, and my heart feels calmer now. I highly recommend it!
8
We examined whether serum magnesium levels at admission could predict in-hospital outcomes for patients experiencing a heart attack.
In our study of 259 acute myocardial infarction patients, those with lower magnesium levels (below 1.82 mg/dl) faced significantly higher risks of complications, including arrhythmias and even death, compared to those with healthier levels.
Our findings suggest that maintaining adequate magnesium levels is essential for better health outcomes after a heart attack.
In our study of 259 acute myocardial infarction patients, those with lower magnesium levels (below 1.82 mg/dl) faced significantly higher risks of complications, including arrhythmias and even death, compared to those with healthier levels.
Our findings suggest that maintaining adequate magnesium levels is essential for better health outcomes after a heart attack.
7
We investigated how variations in serum magnesium levels might relate to in-hospital mortality for patients experiencing an acute myocardial infarction (AMI), commonly known as a heart attack. The research examined data from over 2,700 patients in intensive care, allowing us to analyze trends over time.
What we found was significant: elevated magnesium levels at ICU admission and changes in magnesium levels within the first 48 hours of ICU stay were associated with a higher risk of in-hospital mortality among these patients. In fact, 18.6% of the patients in our study did not survive their hospital stay.
This relationship remained consistent across different groups, regardless of age, gender, or health severity scores. It highlights the importance of closely monitoring magnesium levels during crucial treatment periods as it may provide insight that could help tailor treatments to improve patient outcomes.
However, we must note that while the study shows an association, it does not isolate the effects of magnesium treatment itself. This means we can't definitively say that magnesium treatment alone drastically alters mortality rates after a heart attack. Instead, the findings suggest magnesium monitoring is vital in intensive care settings.
What we found was significant: elevated magnesium levels at ICU admission and changes in magnesium levels within the first 48 hours of ICU stay were associated with a higher risk of in-hospital mortality among these patients. In fact, 18.6% of the patients in our study did not survive their hospital stay.
This relationship remained consistent across different groups, regardless of age, gender, or health severity scores. It highlights the importance of closely monitoring magnesium levels during crucial treatment periods as it may provide insight that could help tailor treatments to improve patient outcomes.
However, we must note that while the study shows an association, it does not isolate the effects of magnesium treatment itself. This means we can't definitively say that magnesium treatment alone drastically alters mortality rates after a heart attack. Instead, the findings suggest magnesium monitoring is vital in intensive care settings.
References
- Demirtola Aİ, Mammadli A, Çiçek G. The Role of Magnesium Levels in the Progression of Contrast-Induced Nephropathy in Patients With STEMI Undergoing Primary PCI. Angiology. 2025. 10.1177/00033197251314629
- Jin Y, Lin Q, Wang D, Gong M, Huang W, et al. Hypomagnesemia is a Risk Factor for Acute Kidney Injury in Patients Admitted With ST-Segment Elevation Myocardial Infarction: A Retrospective Observational Study. J Ren Nutr. 2024. 10.1053/j.jrn.2024.12.006
- Naveen V, Rajesh Lenin R, Stanley LM, Kumar JS. Serum Magnesium Levels and QTc Interval Prolongation As Prognostic Markers in Acute Myocardial Infarction: A Randomized Controlled Study. Cureus. 2024;16:e66051. 10.7759/cureus.66051
- Zhuang J, Zhang Q, Wang H, Su PH, Chen PY. Association between short-term changes in serum magnesium and in-hospital mortality following acute myocardial infarction: a cohort study based on the MIMIC database. Magnes Res. 2024;37:45. 10.1684/mrh.2024.0517
- Zhang J, Sun D, Guo Y, Tong J, Liu Q, et al. Targeted delivery of black phosphorus nanosheets by ROS responsive complex hydrogel based on angiogenesis and antioxidant promotes myocardial infarction repair. J Nanobiotechnology. 2024;22:433. 10.1186/s12951-024-02685-0
- Wang B, Pan S, Nie C, Zou R, Liu J, et al. Magnesium implantation as a continuous hydrogen production generator for the treatment of myocardial infarction in rats. Sci Rep. 2024;14:10959. 10.1038/s41598-024-60609-2
- Fan C, Qin K, Iroegbu CD, Xiang K, Gong Y, et al. Magnesium lithospermate B enhances the potential of human-induced pluripotent stem cell-derived cardiomyocytes for myocardial repair. Chin Med J (Engl). 2024;137:1857. 10.1097/CM9.0000000000002867
- Segev A, Shechter M, Tsur AM, Belkin D, Cohen H, et al. Serum Magnesium Is Associated with Long-Term Survival of Non-ST-Elevation Myocardial Infarction Patients. Nutrients. 2023;15. 10.3390/nu15194299
- Włodarczak A, Rola P, Barycki M, Furtan Ł, Łanocha M, et al. Mid-term safety and efficacy of magnesium bioresorbable vascular scaffolds - magmaris in diabetic population. 2-Years outcome in acute coronary syndrome cohort. Diab Vasc Dis Res. 2023;20:14791641231188705. 10.1177/14791641231188705
- Sabah Z, Wani J, Deajim M, Al Zomia AS, Asiri A, et al. Serum Magnesium in Patients With Acute Myocardial Infarction and Its Effect on Cardiac Complications and Mortality in Myocardial Infarction Patients. Cureus. 2023;15:e38147. 10.7759/cureus.38147
- An P, Wan S, Luo Y, Luo J, Zhang X, et al. Micronutrient Supplementation to Reduce Cardiovascular Risk. J Am Coll Cardiol. 2022;80:2269. 10.1016/j.jacc.2022.09.048
- Evers I, Cruijsen E, Kornaat I, Winkels RM, Busstra MC, et al. Dietary magnesium and risk of cardiovascular and all-cause mortality after myocardial infarction: A prospective analysis in the Alpha Omega Cohort. Front Cardiovasc Med. 2022;9:936772. 10.3389/fcvm.2022.936772
- Rola P, Włodarczak A, Łanocha M, Barycki M, Szudrowicz M, et al. Outcomes of the two generations of bioresorbable scaffolds (Magmaris vs. Absorb) in acute coronary syndrome in routine clinical practice. Cardiol J. 2022;30:870. 10.5603/CJ.a2022.0047
- Ortega-Paz L, Brugaletta S, Gomez-Lara J, Alfonso F, Cequier A, et al. Magnesium-based resorbable scaffold vs permanent metallic sirolimus-eluting stent in patients with ST-segment elevation myocardial infarction: 3-year results of the MAGSTEMI randomised controlled trial. EuroIntervention. 2022;18:e389. 10.4244/EIJ-D-21-00651
- Hasan MZ, Nahar K, Sharif JU, Paul SC, Thakur AK, et al. Serum Magnesium Level and It's Relation in Predicting Adverse In-Hospital Outcome in Patients with First Attack of Myocardial Infarction. Mymensingh Med J. 2023;32:65. PubMed
- Fallesen CO, Antonsen L, Maehara A, Noori M, Hougaard M, et al. Optical Coherence Tomography- Versus Angiography-Guided Magnesium Bioresorbable Scaffold Implantation in NSTEMI Patients. Cardiovasc Revasc Med. 2022;40:101. 10.1016/j.carrev.2021.12.003
- Włodarczak A, Łanocha M, Lesiak M, Benett J, Szudrowicz M, et al. Long-term clinical follow-up of the resorbable magnesium scaffolds in acute coronary syndrome patients. Kardiol Pol. 2021;79:827. 10.33963/KP.a2021.0035
- Açıkgöz E, Açıkgöz SK, Çiçek G. Serum magnesium concentration may predict no-reflow phenomenon in primary angioplasty for ST-elevation myocardial infarction. Magnes Res. 2020;33:123. 10.1684/mrh.2021.0477
- Shlezinger M, Amitai Y, Goldenberg I, Atar S, Shechter M. Acute myocardial infarction severity, complications, and mortality associated with lack of magnesium intake through consumption of desalinated seawater. Magnes Res. 2019;32:39. 10.1684/mrh.2019.0449
- Sabaté M, Alfonso F, Cequier A, Romaní S, Bordes P, et al. Magnesium-Based Resorbable Scaffold Versus Permanent Metallic Sirolimus-Eluting Stent in Patients With ST-Segment Elevation Myocardial Infarction: The MAGSTEMI Randomized Clinical Trial. Circulation. 2019;140:1904. 10.1161/CIRCULATIONAHA.119.043467
- Ielasi A, Cerrato E, Geraci S, Campo G, Garro N, et al. Sirolimus-Eluting Magnesium Resorbable Scaffold Implantation in Patients with Acute Myocardial Infarction. Cardiology. 2019;142:93. 10.1159/000499536
- Lee TM, Chang NC, Lin SZ. Effect of proton pump inhibitors on sympathetic hyperinnervation in infarcted rats: Role of magnesium. PLoS One. 2018;13:e0202979. 10.1371/journal.pone.0202979
- de Hemptinne Q, Picard F, Briki R, Awada A, Silance PG, et al. Drug-Eluting Resorbable Magnesium Scaffold Implantation in ST-Segment Elevation Myocardial Infarction: A Pilot Study. J Invasive Cardiol. 2018;30:202. PubMed
- Wang XQ, Yin S, Wang QW, Bai WW, Tan RH, et al. Vitamin B6 allosterically activates AMPK to promote postischemic angiogenesis in mice. Eur J Pharmacol. 2025;993:177413. 10.1016/j.ejphar.2025.177413
- Evens L, Beliën H, D'Haese S, Haesen S, Verboven M, et al. Combinational Therapy of Cardiac Atrial Appendage Stem Cells and Pyridoxamine: The Road to Cardiac Repair?. Int J Mol Sci. 2021;22. 10.3390/ijms22179266
- Hodzic E. Potential Anti-Inflammatory Treatment of Ischemic Heart Disease. Med Arch. 2018;72:94. 10.5455/medarh.2018.72.94-98
- Deluyker D, Ferferieva V, Driesen RB, Verboven M, Lambrichts I, et al. Pyridoxamine improves survival and limits cardiac dysfunction after MI. Sci Rep. 2017;7:16010. 10.1038/s41598-017-16255-y
- Ding YP, Pedersen EK, Johansson S, Gregory JF, Ueland PM, et al. B vitamin treatments modify the risk of myocardial infarction associated with a MTHFD1 polymorphism in patients with stable angina pectoris. Nutr Metab Cardiovasc Dis. 2016;26:495. 10.1016/j.numecd.2015.12.009
- Myung SK, Ju W, Cho B, Oh SW, Park SM, et al. Efficacy of vitamin and antioxidant supplements in prevention of cardiovascular disease: systematic review and meta-analysis of randomised controlled trials. BMJ. 2013;346:f10. 10.1136/bmj.f10
Data last updated:
