Reviewed By
Overview
SCIENTIFIC SCORE
Moderately Effective
Based on 16 Researches
8
USERS' SCORE
Good
Based on 5 Reviews
8.4
Supplement Facts
Serving Size: 1 MicroLingual® Tablet
Amount Per Serving
%DV
Vitamin D (as cholecalciferol)
125 mcg (5,000 IU)
625%
Top Medical Research Studies
We explored the effects of vitamin D supplementation in patients with liver cirrhosis—a progressive disease that often manifests alongside vitamin D deficiency. In a carefully designed study, we involved sixty patients who participated in a double-blind, randomized controlled trial. Each patient received either a weekly dose of 50,000 IU of vitamin D or a placebo over 12 weeks.
Throughout the study, we assessed key health markers before and after supplementation, including liver function tests, lipid profiles, and glycaemic indices such as fasting blood glucose and insulin resistance. By the end of the trial, our findings indicated that vitamin D supplementation significantly improved fasting blood glucose levels and insulin resistance in the participants.
Specifically, we observed noteworthy increases in serum 25-hydroxy-vitamin D levels and reductions in fasting blood glucose and insulin resistance indicators. These results suggest that vitamin D might be an important player in improving metabolic health for those suffering from liver cirrhosis.
Throughout the study, we assessed key health markers before and after supplementation, including liver function tests, lipid profiles, and glycaemic indices such as fasting blood glucose and insulin resistance. By the end of the trial, our findings indicated that vitamin D supplementation significantly improved fasting blood glucose levels and insulin resistance in the participants.
Specifically, we observed noteworthy increases in serum 25-hydroxy-vitamin D levels and reductions in fasting blood glucose and insulin resistance indicators. These results suggest that vitamin D might be an important player in improving metabolic health for those suffering from liver cirrhosis.
Read More
9
We explored the impact of vitamin D on metabolic associated fatty liver disease (MAFLD) by examining both animal and cell models. Our study involved male C57BL/6J mice, which were put on a high-fat diet and then treated with vitamin D for 16 weeks. At the same time, we analyzed liver cells that were exposed to palmitic acid to mimic fatty liver conditions.
Our findings revealed that vitamin D not only helped reduce body weight and improve liver health, but it also played a role in restoring normal metabolic functions. By reducing inflammation and protecting liver cells from damage, vitamin D enhanced insulin sensitivity and affected fat metabolism positively.
Notably, we found that vitamin D helped inhibit a harmful process known as ferroptosis—a type of cell death linked to liver injury. Through various assays, we confirmed that vitamin D treatment boosted the liver's antioxidant capabilities and lessened iron buildup in liver cells. Overall, our research suggests that vitamin D could be a promising therapeutic option for individuals suffering from MAFLD.
Our findings revealed that vitamin D not only helped reduce body weight and improve liver health, but it also played a role in restoring normal metabolic functions. By reducing inflammation and protecting liver cells from damage, vitamin D enhanced insulin sensitivity and affected fat metabolism positively.
Notably, we found that vitamin D helped inhibit a harmful process known as ferroptosis—a type of cell death linked to liver injury. Through various assays, we confirmed that vitamin D treatment boosted the liver's antioxidant capabilities and lessened iron buildup in liver cells. Overall, our research suggests that vitamin D could be a promising therapeutic option for individuals suffering from MAFLD.
Read More
9.5
We designed and synthesized a series of 37 non-steroidal compounds aimed at activating the vitamin D receptor (VDR) to explore their potential in treating liver fibrosis. This condition involves an unhealthy buildup of fibrous tissue in the liver, often leading to serious complications.
Our research found that more than a third of these novel compounds displayed strong affinity for VDR and showed the ability to activate it. Among these, one compound, E15, stood out as particularly effective. It significantly inhibited the activation of hepatic stellate cells (HSCs), which play a critical role in the progression of liver fibrosis, thereby reducing the production of harmful extracellular matrix components.
Encouraged by these promising in vitro results, we proceeded to test E15 in a mouse model of liver fibrosis induced by carbon tetrachloride. The results were remarkable; E15 not only decreased fibrosis and collagen deposition, but also improved liver function without the negative side effect of hypercalcemia, which is often associated with traditional VDR agonists.
These findings suggest that E15 could be a powerful and safer alternative for addressing liver fibrosis, highlighting the significant therapeutic potential of targeting the vitamin D receptor in liver diseases.
Our research found that more than a third of these novel compounds displayed strong affinity for VDR and showed the ability to activate it. Among these, one compound, E15, stood out as particularly effective. It significantly inhibited the activation of hepatic stellate cells (HSCs), which play a critical role in the progression of liver fibrosis, thereby reducing the production of harmful extracellular matrix components.
Encouraged by these promising in vitro results, we proceeded to test E15 in a mouse model of liver fibrosis induced by carbon tetrachloride. The results were remarkable; E15 not only decreased fibrosis and collagen deposition, but also improved liver function without the negative side effect of hypercalcemia, which is often associated with traditional VDR agonists.
These findings suggest that E15 could be a powerful and safer alternative for addressing liver fibrosis, highlighting the significant therapeutic potential of targeting the vitamin D receptor in liver diseases.
Read More
Most Useful Reviews
9
Gentle on liver
The best form of vitamin D! It does not stress the liver and works quickly through the mucous membranes. My family, including the youngest, enjoys the slightly sweet taste and benefits.
Read More
9
Potent sublingual form
Lingual vitamin D3 is remarkably effective, dissolving under the tongue for rapid absorption into the bloodstream, bypassing digestion. It increased my vitamin D levels, something no other drugs could do due to my autoimmune condition. It’s sweet and convenient for all ages.
Read More
7.5
Vitamin D improvement
5 people found this helpful
Good remedy for my vitamin D deficiency. After having liver disease, my body was weak, and tests showed several deficiencies. A standard pharmacy vitamin D did not help, but when I switched to MicroLingual sublingual vitamin D, I saw improvement. I hope this will help me eliminate my vitamin D deficiency.
Read More
Medical Researches
SCIENTIFIC SCORE
Moderately Effective
Based on 16 Researches
8
- All Researches
9.5
We designed and synthesized a series of 37 non-steroidal compounds aimed at activating the vitamin D receptor (VDR) to explore their potential in treating liver fibrosis. This condition involves an unhealthy buildup of fibrous tissue in the liver, often leading to serious complications.
Our research found that more than a third of these novel compounds displayed strong affinity for VDR and showed the ability to activate it. Among these, one compound, E15, stood out as particularly effective. It significantly inhibited the activation of hepatic stellate cells (HSCs), which play a critical role in the progression of liver fibrosis, thereby reducing the production of harmful extracellular matrix components.
Encouraged by these promising in vitro results, we proceeded to test E15 in a mouse model of liver fibrosis induced by carbon tetrachloride. The results were remarkable; E15 not only decreased fibrosis and collagen deposition, but also improved liver function without the negative side effect of hypercalcemia, which is often associated with traditional VDR agonists.
These findings suggest that E15 could be a powerful and safer alternative for addressing liver fibrosis, highlighting the significant therapeutic potential of targeting the vitamin D receptor in liver diseases.
Our research found that more than a third of these novel compounds displayed strong affinity for VDR and showed the ability to activate it. Among these, one compound, E15, stood out as particularly effective. It significantly inhibited the activation of hepatic stellate cells (HSCs), which play a critical role in the progression of liver fibrosis, thereby reducing the production of harmful extracellular matrix components.
Encouraged by these promising in vitro results, we proceeded to test E15 in a mouse model of liver fibrosis induced by carbon tetrachloride. The results were remarkable; E15 not only decreased fibrosis and collagen deposition, but also improved liver function without the negative side effect of hypercalcemia, which is often associated with traditional VDR agonists.
These findings suggest that E15 could be a powerful and safer alternative for addressing liver fibrosis, highlighting the significant therapeutic potential of targeting the vitamin D receptor in liver diseases.
Read More
9
We set out to understand how vitamin D might influence liver disease, particularly non-alcoholic fatty liver disease (NAFLD) related to obesity. For our research, we used a high-fat diet to create a mouse model that mimics the condition. We then supplemented these mice with vitamin D via injections over several weeks to see if it could alleviate the liver issues associated with their diet.
Our findings revealed that a high-fat diet led to vitamin D deficiency, insulin resistance, and a notable increase in liver weight. This also resulted in elevated liver enzymes and triglyceride levels, contributing to steatohepatitis, a concerning liver condition. When we introduced vitamin D supplementation, we observed a significant recovery in liver weight and overall improvement in liver health. The treatment also lowered harmful enzymes and triglycerides while helping control markers related to inflammation and fibrosis.
This study highlights that vitamin D supplementation can positively impact liver health in cases linked to obesity, offering an accessible and potentially effective strategy for addressing NAFLD. Notably, we found no adverse effects from the vitamin D treatment, suggesting it could be a safe option for individuals at risk.
Our findings revealed that a high-fat diet led to vitamin D deficiency, insulin resistance, and a notable increase in liver weight. This also resulted in elevated liver enzymes and triglyceride levels, contributing to steatohepatitis, a concerning liver condition. When we introduced vitamin D supplementation, we observed a significant recovery in liver weight and overall improvement in liver health. The treatment also lowered harmful enzymes and triglycerides while helping control markers related to inflammation and fibrosis.
This study highlights that vitamin D supplementation can positively impact liver health in cases linked to obesity, offering an accessible and potentially effective strategy for addressing NAFLD. Notably, we found no adverse effects from the vitamin D treatment, suggesting it could be a safe option for individuals at risk.
Read More
We explored the effects of vitamin D supplementation in patients with liver cirrhosis—a progressive disease that often manifests alongside vitamin D deficiency. In a carefully designed study, we involved sixty patients who participated in a double-blind, randomized controlled trial. Each patient received either a weekly dose of 50,000 IU of vitamin D or a placebo over 12 weeks.
Throughout the study, we assessed key health markers before and after supplementation, including liver function tests, lipid profiles, and glycaemic indices such as fasting blood glucose and insulin resistance. By the end of the trial, our findings indicated that vitamin D supplementation significantly improved fasting blood glucose levels and insulin resistance in the participants.
Specifically, we observed noteworthy increases in serum 25-hydroxy-vitamin D levels and reductions in fasting blood glucose and insulin resistance indicators. These results suggest that vitamin D might be an important player in improving metabolic health for those suffering from liver cirrhosis.
Throughout the study, we assessed key health markers before and after supplementation, including liver function tests, lipid profiles, and glycaemic indices such as fasting blood glucose and insulin resistance. By the end of the trial, our findings indicated that vitamin D supplementation significantly improved fasting blood glucose levels and insulin resistance in the participants.
Specifically, we observed noteworthy increases in serum 25-hydroxy-vitamin D levels and reductions in fasting blood glucose and insulin resistance indicators. These results suggest that vitamin D might be an important player in improving metabolic health for those suffering from liver cirrhosis.
Read More
9
We explored the relationship between vitamin D and bile duct health, particularly focusing on biliary atresia (BA), a condition that leads to bile duct obstruction in children. Our research centered on vitamin D's receptor (VDR) and its role in protecting bile duct epithelial cells from damage caused by viruses, specifically double-stranded RNA viruses.
Through a combination of laboratory and animal studies, we assessed the expression of VDR in bile duct cells from pediatric patients, noting its connection to cholangitis rates after treatment. We discovered that activating VDR with vitamin D3 significantly reduced cell damage and apoptosis, which is the process of programmed cell death that can worsen BA.
We found that vitamin D3 helped mitigate viral-induced inflammation and cell death through specific cellular pathways, including one called the PLA2/PKC/ERK pathway. This suggests that vitamin D could be a valuable therapeutic option in managing liver diseases like BA, potentially offering new avenues for treatment and patient care.
Through a combination of laboratory and animal studies, we assessed the expression of VDR in bile duct cells from pediatric patients, noting its connection to cholangitis rates after treatment. We discovered that activating VDR with vitamin D3 significantly reduced cell damage and apoptosis, which is the process of programmed cell death that can worsen BA.
We found that vitamin D3 helped mitigate viral-induced inflammation and cell death through specific cellular pathways, including one called the PLA2/PKC/ERK pathway. This suggests that vitamin D could be a valuable therapeutic option in managing liver diseases like BA, potentially offering new avenues for treatment and patient care.
Read More
9
We explored the impact of vitamin D on metabolic associated fatty liver disease (MAFLD) by examining both animal and cell models. Our study involved male C57BL/6J mice, which were put on a high-fat diet and then treated with vitamin D for 16 weeks. At the same time, we analyzed liver cells that were exposed to palmitic acid to mimic fatty liver conditions.
Our findings revealed that vitamin D not only helped reduce body weight and improve liver health, but it also played a role in restoring normal metabolic functions. By reducing inflammation and protecting liver cells from damage, vitamin D enhanced insulin sensitivity and affected fat metabolism positively.
Notably, we found that vitamin D helped inhibit a harmful process known as ferroptosis—a type of cell death linked to liver injury. Through various assays, we confirmed that vitamin D treatment boosted the liver's antioxidant capabilities and lessened iron buildup in liver cells. Overall, our research suggests that vitamin D could be a promising therapeutic option for individuals suffering from MAFLD.
Our findings revealed that vitamin D not only helped reduce body weight and improve liver health, but it also played a role in restoring normal metabolic functions. By reducing inflammation and protecting liver cells from damage, vitamin D enhanced insulin sensitivity and affected fat metabolism positively.
Notably, we found that vitamin D helped inhibit a harmful process known as ferroptosis—a type of cell death linked to liver injury. Through various assays, we confirmed that vitamin D treatment boosted the liver's antioxidant capabilities and lessened iron buildup in liver cells. Overall, our research suggests that vitamin D could be a promising therapeutic option for individuals suffering from MAFLD.
Read More
User Reviews
USERS' SCORE
Good
Based on 5 Reviews
8.4
- All Reviews
- Positive Reviews
- Negative Reviews
9
Gentle on liver
The best form of vitamin D! It does not stress the liver and works quickly through the mucous membranes. My family, including the youngest, enjoys the slightly sweet taste and benefits.
Read More
9
Potent sublingual form
Lingual vitamin D3 is remarkably effective, dissolving under the tongue for rapid absorption into the bloodstream, bypassing digestion. It increased my vitamin D levels, something no other drugs could do due to my autoimmune condition. It’s sweet and convenient for all ages.
Read More
7.5
Vitamin D improvement
5 people found this helpful
Good remedy for my vitamin D deficiency. After having liver disease, my body was weak, and tests showed several deficiencies. A standard pharmacy vitamin D did not help, but when I switched to MicroLingual sublingual vitamin D, I saw improvement. I hope this will help me eliminate my vitamin D deficiency.
Read More
7.5
Effective treatment option
5 people found this helpful
Only this vitamin D works for me! I spent nine months trying to raise my vitamin D levels with capsules but saw little result. This sublingual form finally showed results in my tests! I need a higher dose due to a genetic issue affecting my VDR gene after liver disease. Now it’s easy to take and has improved my vitamin D levels significantly.
Read More
9
Rapid absorption
Perfectly effective! The best form of vitamin D for quickly raising levels, especially if you have liver issues. It dissolves instantly on the tongue and is easily absorbed, with a pleasant taste.
Read More
Frequently Asked Questions
7.5
Effective treatment option
5 people found this helpful
Only this vitamin D works for me! I spent nine months trying to raise my vitamin D levels with capsules but saw little result. This sublingual form finally showed results in my tests! I need a higher dose due to a genetic issue affecting my VDR gene after liver disease. Now it’s easy to take and has improved my vitamin D levels significantly.
7.5
Vitamin D improvement
5 people found this helpful
Good remedy for my vitamin D deficiency. After having liver disease, my body was weak, and tests showed several deficiencies. A standard pharmacy vitamin D did not help, but when I switched to MicroLingual sublingual vitamin D, I saw improvement. I hope this will help me eliminate my vitamin D deficiency.
9
Rapid absorption
Perfectly effective! The best form of vitamin D for quickly raising levels, especially if you have liver issues. It dissolves instantly on the tongue and is easily absorbed, with a pleasant taste.
9
Potent sublingual form
Lingual vitamin D3 is remarkably effective, dissolving under the tongue for rapid absorption into the bloodstream, bypassing digestion. It increased my vitamin D levels, something no other drugs could do due to my autoimmune condition. It’s sweet and convenient for all ages.
9
Gentle on liver
The best form of vitamin D! It does not stress the liver and works quickly through the mucous membranes. My family, including the youngest, enjoys the slightly sweet taste and benefits.
9
We set out to understand how vitamin D might influence liver disease, particularly non-alcoholic fatty liver disease (NAFLD) related to obesity. For our research, we used a high-fat diet to create a mouse model that mimics the condition. We then supplemented these mice with vitamin D via injections over several weeks to see if it could alleviate the liver issues associated with their diet.
Our findings revealed that a high-fat diet led to vitamin D deficiency, insulin resistance, and a notable increase in liver weight. This also resulted in elevated liver enzymes and triglyceride levels, contributing to steatohepatitis, a concerning liver condition. When we introduced vitamin D supplementation, we observed a significant recovery in liver weight and overall improvement in liver health. The treatment also lowered harmful enzymes and triglycerides while helping control markers related to inflammation and fibrosis.
This study highlights that vitamin D supplementation can positively impact liver health in cases linked to obesity, offering an accessible and potentially effective strategy for addressing NAFLD. Notably, we found no adverse effects from the vitamin D treatment, suggesting it could be a safe option for individuals at risk.
Our findings revealed that a high-fat diet led to vitamin D deficiency, insulin resistance, and a notable increase in liver weight. This also resulted in elevated liver enzymes and triglyceride levels, contributing to steatohepatitis, a concerning liver condition. When we introduced vitamin D supplementation, we observed a significant recovery in liver weight and overall improvement in liver health. The treatment also lowered harmful enzymes and triglycerides while helping control markers related to inflammation and fibrosis.
This study highlights that vitamin D supplementation can positively impact liver health in cases linked to obesity, offering an accessible and potentially effective strategy for addressing NAFLD. Notably, we found no adverse effects from the vitamin D treatment, suggesting it could be a safe option for individuals at risk.
We explored the effects of vitamin D supplementation in patients with liver cirrhosis—a progressive disease that often manifests alongside vitamin D deficiency. In a carefully designed study, we involved sixty patients who participated in a double-blind, randomized controlled trial. Each patient received either a weekly dose of 50,000 IU of vitamin D or a placebo over 12 weeks.
Throughout the study, we assessed key health markers before and after supplementation, including liver function tests, lipid profiles, and glycaemic indices such as fasting blood glucose and insulin resistance. By the end of the trial, our findings indicated that vitamin D supplementation significantly improved fasting blood glucose levels and insulin resistance in the participants.
Specifically, we observed noteworthy increases in serum 25-hydroxy-vitamin D levels and reductions in fasting blood glucose and insulin resistance indicators. These results suggest that vitamin D might be an important player in improving metabolic health for those suffering from liver cirrhosis.
Throughout the study, we assessed key health markers before and after supplementation, including liver function tests, lipid profiles, and glycaemic indices such as fasting blood glucose and insulin resistance. By the end of the trial, our findings indicated that vitamin D supplementation significantly improved fasting blood glucose levels and insulin resistance in the participants.
Specifically, we observed noteworthy increases in serum 25-hydroxy-vitamin D levels and reductions in fasting blood glucose and insulin resistance indicators. These results suggest that vitamin D might be an important player in improving metabolic health for those suffering from liver cirrhosis.
8
Our study aimed to investigate how vitamin D supplementation can influence cognitive function and inflammation in patients suffering from decompensated cirrhosis. We recruited patients from two hospitals in Spain and monitored their progress for up to 12 months after starting vitamin D treatment.
Interestingly, we found that the patients who had lower levels of vitamin D at the beginning showed improved cognitive functions, especially in areas like working memory, after supplementation. Moreover, we observed that vitamin D helped reduce markers of inflammation in these patients, suggesting a connection between vitamin D levels and inflammatory responses.
The results indicated not only cognitive improvements but also a clearer understanding of how vitamin D can modulate inflammation in liver disease. This reinforces the idea that maintaining adequate vitamin D levels might be beneficial for those coping with liver conditions, specifically decompensated cirrhosis.
Interestingly, we found that the patients who had lower levels of vitamin D at the beginning showed improved cognitive functions, especially in areas like working memory, after supplementation. Moreover, we observed that vitamin D helped reduce markers of inflammation in these patients, suggesting a connection between vitamin D levels and inflammatory responses.
The results indicated not only cognitive improvements but also a clearer understanding of how vitamin D can modulate inflammation in liver disease. This reinforces the idea that maintaining adequate vitamin D levels might be beneficial for those coping with liver conditions, specifically decompensated cirrhosis.
References
- Chung SI, Liang L, Han H, Park KH, Lee JH, et al. Vitamin D Attenuates Non-Alcoholic Fatty Liver Disease in High-Fat Diet-Induced Obesity Murine Model. Yonsei Med J. 2025;66:75. 10.3349/ymj.2024.0038
- Derogar Kasmaei SR, Parastouei K, Hosseini Ahangar B, Saberifiroozi M, Taghdir M. Effects of vitamin D supplementation on the glycaemic indices, lipid profile and liver function tests in patients with cirrhosis: a double-blind randomised controlled trial. BMJ Nutr Prev Health. 2024;7:e000938. 10.1136/bmjnph-2024-000938
- Liu N, Zhao P, Cao P, Hui J, Pan Y, et al. Vitamin D3/VDR alleviates double-stranded RNA virus -induced biliary epithelial cell damage by inhibiting autophagy. BMC Gastroenterol. 2025;25:44. 10.1186/s12876-025-03640-5
- Zou C, Liu X, He M, Sun Y, Sang Y, et al. Insulin Resistance Mediates the Association Between Vitamin D and Non-Alcoholic Fatty Liver Disease. Int J Prev Med. 2024;15:77. 10.4103/ijpvm.ijpvm_221_23
- Diaz-Ruiz R, Poca M, Roman E, Panadero-Gomez R, Cuyàs B, et al. Vitamin D Supplementation Is Associated with Inflammation Amelioration and Cognitive Improvement in Decompensated Patients with Cirrhosis. Nutrients. 2025;17. 10.3390/nu17020226
- Wang Y, Jin J, Chen S, Shen Y. Modulation of magnesium intake on the association between vitamin D deficiency and severe hepatic steatosis in overweight and obese individuals. Magnes Res. 2024;37:58. 10.1684/mrh.2024.0536
- Jiang R, Lu M, Hua Y, Hong Z. Association between serum vitamin D and depression among non-alcoholic fatty liver disease. Asia Pac J Clin Nutr. 2025;34:112. 10.6133/apjcn.202502_34(1).0011
- Gao F, Guan C, Cheng N, Liu Y, Wu Y, et al. Design, synthesis, and anti-liver fibrosis activity of novel non-steroidal vitamin D receptor agonists based on open-ring steroid scaffold. Eur J Med Chem. 2025;286:117250. 10.1016/j.ejmech.2025.117250
- Biswas SA, Rukunuzzaman M, Biswas RK, Rahman SMH, Alam MS. Serum Vitamin D Status in Infants with Cholestatic Jaundice. Mymensingh Med J. 2025;34:192.
- Munoli AS, Mantur PG, Jalawadi VM. Child-Pugh Score and Vitamin D: Exploring a New Frontier in Liver Cirrhosis Assessment. Cureus. 2024;16:e74738. 10.7759/cureus.74738
- Liang Y, Jiang X, Zhao X, Tang T, Fan X, et al. Vitamin D alleviates HFD-induced hepatic fibrosis by inhibiting DNMT1 to affect the TGFβ1/Smad3 pathway. iScience. 2024;27:111262. 10.1016/j.isci.2024.111262
- Miao Y, Jiang Z, Song H, Zhang Y, Chen H, et al. Vitamin D supplementation alleviates high fat diet-induced metabolic associated fatty liver disease by inhibiting ferroptosis pathway. Eur J Nutr. 2024;64:50. 10.1007/s00394-024-03554-0
- Huang N, Su X, Yu T, Wu X, Lu B, et al. Serum 25-hydroxy vitamin D level is associated with elastography-detected liver fibrosis in patients with type 2 diabetes mellitus in China. Front Endocrinol (Lausanne). 2024;15:1420088. 10.3389/fendo.2024.1420088
- Johnson CD, Stevens CM, Bennett MR, Litch AB, Rodrigue EM, et al. The Role of Vitamin D Deficiency in Hepatic Encephalopathy: A Review of Pathophysiology, Clinical Outcomes, and Therapeutic Potential. Nutrients. 2024;16. 10.3390/nu16234007
- Morsy MA, Abdel-Latif R, Ibrahim MF, Marey H, Abdel-Gaber SA. Calcitriol ameliorates cisplatin-induced hepatorenal toxicity via regulation of Nrf2-Mrp2/p38 MAPK signaling in mice. Int J Immunopathol Pharmacol. 2024;38:3946320241306276. 10.1177/03946320241306276
- Luo WJ, Dong XW, Ye H, Zhao QS, Zhang QB, et al. Vitamin D 1,25-Dihydroxyvitamin D reduces lipid accumulation in hepatocytes by inhibiting M1 macrophage polarization. World J Gastrointest Oncol. 2024;16:4685. 10.4251/wjgo.v16.i12.4685
