' 
SCIENTIFIC SCORE
Moderately Effective
Based on 19 Researches
8.3
USERS' SCORE
Good
Based on 1 Review
8.5
Supplement Facts
Serving Size: 1 Level Scoop (About 10 g)
Amount Per Serving
%DV
Calories
40
Total Fat
1 g
1%¹
Total Carbohydrate
5 g
2%¹
Dietary Fiber
1 g
4%¹
Total Sugars
<1 g
+
Includes 0 g Added Sugars
0%
Protein
3 g
Vitamin A (as beta-carotene)
2,400 mcg
267%
Vitamin C (from Acerola Cherry)
40 mg
44%
Calcium (from Algae)
60 mg
5%
Iron
3 mg
17%
Sodium
85 mg
4%
Perfect Green Juice BlendOrganic Barley Grass (Juice), Organic Alfalfa Grass (Juice), Organic Wheat Grass (Juice), Organic Oat Grass (Juice)
5 g
+
Perfect Protein-Mineral BlendOrganic Spirulina, Rice Bran Solubles, Chlorella, Calcified Red Algae (Lithothamnion corallioides), Kelp Blend.
3.3 g
+
Fermented Whole Food MatrixOrganic Barley Grass, Organic Oat Grass, Organic Wheat Grass, Organic Alfalfa Grass, Organic Flax Seed Sprout, Amaranth Sprout, Chia Seed Sprout, Garbanzo Bean Sprout, Organic Sesame Seed Sprout, Quinoa Sprout, Organic Buckwheat Sprout, Millet Sprout, Sunflower Seed Sprout, Adzuki Bean Sprout, Organic Pumpkin Seed Sprout, Lentil Sprout
1.2 mg
+
Acerola Cherry Extract (Fruit)
300 mg
+
Perfect Veggie Juice BlendOrganic Alfalfa Grass, Organic Beet (Root), Organic Carrot (Root), Organic Broccoli (Flower & Stem), Organic Tomato (Fruit), Cucumber (Gourd), Organic Kale (Leaf), Organic Spinach (Leaf), Organic Parsley (Leaf), Organic Green Cabbage (Leaf), Celery (Stalk), Cauliflower (Flower & Stem), Organic Green Bell Pepper (Fruit), Asparagus (Flower & Stem), Brussels Sprout (Leaf), Organic Onion (Bulb), Organic Garlic (Bulb), Organic Ginger (Root)
200 mg
+
Probiotic BlendLactobacillus plantarum, Bifidobacterium longum, Bifidobacterium lactis, Bifidobacterium bifidum, Lactobacillus rhamnosus, Bifidobacterium breve, Lactobacillus paracasei, Lactobacillus casei, Lactobacillus salivarius, Lactobacillus acidophilus (1 Billion CFU at time of manufacture)
6.75 mg
+
Top Medical Research Studies
9
atRA improves stroke outcomes
All trans-retinoic acid protects against acute ischemic stroke by modulating neutrophil functions through STAT1 signaling.
Highly relevant stroke therapy research
We explored how all-trans retinoic acid (atRA), a powerful vitamin A derivative, impacts recovery after an ischemic stroke. In our examination, mice were treated with atRA before and after inducing a stroke through transient middle cerebral artery occlusion (tMCAO). This allowed us to assess not just stroke outcomes, but also how atRA influences inflammation and neutrophil activity in the brain.
Our findings revealed that atRA significantly reduced both the size of brain damage and neurological issues one day after the stroke. Specifically, we observed that treatment with atRA helped to lower neutrophil accumulation in the stroke-affected areas. This is important because an excess of neutrophils can contribute to further damage in the brain post-stroke.
Furthermore, atRA treatment appeared to promote the “N2” phenotype of neutrophils, which is associated with protective functions, and inhibited the formation of harmful neutrophil extracellular traps (NETs). Interestingly, we found that the positive outcomes from atRA were related to a reduction in STAT1 signaling, which plays a role in neutrophil regulation. Even when treatment was started right after the stroke, atRA still offered protective benefits.
In summary, our study shows that atRA can effectively reduce brain inflammation and damage after a stroke, presenting a potential avenue for stroke therapy that warrants further investigation.
Our findings revealed that atRA significantly reduced both the size of brain damage and neurological issues one day after the stroke. Specifically, we observed that treatment with atRA helped to lower neutrophil accumulation in the stroke-affected areas. This is important because an excess of neutrophils can contribute to further damage in the brain post-stroke.
Furthermore, atRA treatment appeared to promote the “N2” phenotype of neutrophils, which is associated with protective functions, and inhibited the formation of harmful neutrophil extracellular traps (NETs). Interestingly, we found that the positive outcomes from atRA were related to a reduction in STAT1 signaling, which plays a role in neutrophil regulation. Even when treatment was started right after the stroke, atRA still offered protective benefits.
In summary, our study shows that atRA can effectively reduce brain inflammation and damage after a stroke, presenting a potential avenue for stroke therapy that warrants further investigation.
Read More
8
Vitamin A reduces stroke risk
Association of vitamin A and its organic compounds with stroke - a systematic review and meta-analysis.
Direct evaluation of vitamin A
We set out to explore how vitamin A and its organic compounds might influence the risk of stroke and stroke-related mortality. Our study examined numerous relevant articles from major databases like PubMed, Scopus, and Web of Science, focusing on human samples for our analyses.
After reviewing 21 studies that included a total of 5,789 stroke patients, we found some intriguing results. The data indicate a notable inverse relationship between vitamin A, particularly retinol and beta-carotene, and stroke risk. Specifically, those with low levels of these compounds had an increased likelihood of both experiencing a stroke and facing higher mortality rates associated with stroke.
While our findings are promising, we must emphasize that variability among the studies suggests that more research is necessary to fully understand the clinical implications of these associations. Insufficiency of retinol and beta-carotene clearly elevates stroke risk, reinforcing the need to maintain adequate levels for potential protective benefits.
After reviewing 21 studies that included a total of 5,789 stroke patients, we found some intriguing results. The data indicate a notable inverse relationship between vitamin A, particularly retinol and beta-carotene, and stroke risk. Specifically, those with low levels of these compounds had an increased likelihood of both experiencing a stroke and facing higher mortality rates associated with stroke.
While our findings are promising, we must emphasize that variability among the studies suggests that more research is necessary to fully understand the clinical implications of these associations. Insufficiency of retinol and beta-carotene clearly elevates stroke risk, reinforcing the need to maintain adequate levels for potential protective benefits.
Read More
9
Vitamin A improves stroke recovery
Neuroprotective effects of all-trans-retinoic acid are mediated via downregulation of TLR4/NF-κB signaling in a rat model of middle cerebral artery occlusion.
Direct focus on vitamin A treatment
We investigated how all-trans-retinoic acid (ATRA), a form of vitamin A, impacts the inflammatory response after a stroke. Using a rat model, we subjected the animals to a condition that simulates a stroke and then treated them with ATRA at two different times after the event. Our goal was to see if ATRA could improve recovery and reduce inflammation in the brain.
The results were promising. We observed that ATRA treatment led to a significant reduction in both the volume of brain damage and the severity of neurological deficits in the rats. This suggests that ATRA helps the brain heal faster after a stroke. Additionally, ATRA seemed to reduce swelling in the brain and lower levels of harmful inflammatory substances released during the recovery process.
Importantly, we noted that ATRA encouraged certain brain cells, known as microglia, to adopt a more protective role rather than a damaging one. This shift indicates that ATRA may impact the brain's healing process by downregulating inflammation via a specific signaling pathway. Overall, our findings support the idea that ATRA could be a potential treatment for reducing inflammation and enhancing recovery after a stroke.
The results were promising. We observed that ATRA treatment led to a significant reduction in both the volume of brain damage and the severity of neurological deficits in the rats. This suggests that ATRA helps the brain heal faster after a stroke. Additionally, ATRA seemed to reduce swelling in the brain and lower levels of harmful inflammatory substances released during the recovery process.
Importantly, we noted that ATRA encouraged certain brain cells, known as microglia, to adopt a more protective role rather than a damaging one. This shift indicates that ATRA may impact the brain's healing process by downregulating inflammation via a specific signaling pathway. Overall, our findings support the idea that ATRA could be a potential treatment for reducing inflammation and enhancing recovery after a stroke.
Read More
Most Useful Reviews
8.8
Improved health condition
Following my mother's stroke on her 62nd birthday and my father's at 58, I was diagnosed with vascular disease at 37. After struggling to change my diet, I began a homeopathic regimen of three pills daily. Six months later, I was able to stop my blood pressure medication, and my health has significantly improved, as has my skin. My doctor supports my lifestyle changes, and I now feel healthier internally.
Read More
Medical Researches
SCIENTIFIC SCORE
Moderately Effective
Based on 19 Researches
8.3
- All Researches
9
Vitamin A improves stroke recovery
Neuroprotective effects of all-trans-retinoic acid are mediated via downregulation of TLR4/NF-κB signaling in a rat model of middle cerebral artery occlusion.
Direct focus on vitamin A treatment
We investigated how all-trans-retinoic acid (ATRA), a form of vitamin A, impacts the inflammatory response after a stroke. Using a rat model, we subjected the animals to a condition that simulates a stroke and then treated them with ATRA at two different times after the event. Our goal was to see if ATRA could improve recovery and reduce inflammation in the brain.
The results were promising. We observed that ATRA treatment led to a significant reduction in both the volume of brain damage and the severity of neurological deficits in the rats. This suggests that ATRA helps the brain heal faster after a stroke. Additionally, ATRA seemed to reduce swelling in the brain and lower levels of harmful inflammatory substances released during the recovery process.
Importantly, we noted that ATRA encouraged certain brain cells, known as microglia, to adopt a more protective role rather than a damaging one. This shift indicates that ATRA may impact the brain's healing process by downregulating inflammation via a specific signaling pathway. Overall, our findings support the idea that ATRA could be a potential treatment for reducing inflammation and enhancing recovery after a stroke.
The results were promising. We observed that ATRA treatment led to a significant reduction in both the volume of brain damage and the severity of neurological deficits in the rats. This suggests that ATRA helps the brain heal faster after a stroke. Additionally, ATRA seemed to reduce swelling in the brain and lower levels of harmful inflammatory substances released during the recovery process.
Importantly, we noted that ATRA encouraged certain brain cells, known as microglia, to adopt a more protective role rather than a damaging one. This shift indicates that ATRA may impact the brain's healing process by downregulating inflammation via a specific signaling pathway. Overall, our findings support the idea that ATRA could be a potential treatment for reducing inflammation and enhancing recovery after a stroke.
Read More
9
Vitamin A aids recovery post-stroke
Retinoic acid alleviates the reduction of Akt and Bad phosphorylation and regulates Bcl-2 family protein interactions in animal models of ischemic stroke.
Direct relevance to stroke treatment
We wanted to understand how vitamin A, specifically its active form known as retinoic acid, can influence recovery from ischemic stroke. In our study, we tested this by administering retinoic acid to animal models before inducing a stroke through middle cerebral artery occlusion.
Our findings were encouraging. We observed that retinoic acid not only reduced neurological deficits but also improved overall brain health after the stroke. The treatment seemed to keep key proteins involved in cell survival—like Akt and Bad—active, which are typically reduced during stroke damage. Notably, retinoic acid also boosted protective protein interactions that prevent cell death, demonstrating how it can support brain resilience in the face of stroke.
Overall, it seems retinoic acid holds promise as a neuroprotective agent that may mitigate the harmful effects of ischemic stroke. This research suggests further exploration of vitamin A in stroke treatments could be beneficial for improving patient outcomes.
Our findings were encouraging. We observed that retinoic acid not only reduced neurological deficits but also improved overall brain health after the stroke. The treatment seemed to keep key proteins involved in cell survival—like Akt and Bad—active, which are typically reduced during stroke damage. Notably, retinoic acid also boosted protective protein interactions that prevent cell death, demonstrating how it can support brain resilience in the face of stroke.
Overall, it seems retinoic acid holds promise as a neuroprotective agent that may mitigate the harmful effects of ischemic stroke. This research suggests further exploration of vitamin A in stroke treatments could be beneficial for improving patient outcomes.
Read More
9
Vitamin A's neuroprotective role
Retinoic Acid-Loaded Nanoparticles Promote Neurovascular Protection in Stroke.
High relevance to stroke treatment
We aimed to understand how vitamin A impacts stroke recovery, specifically by exploring the effects of retinoic acid-loaded nanoparticles. This innovative approach was designed to assess neurovascular protection during stroke events. By administering these nanoparticles, we sought to determine whether they could enhance recovery outcomes for patients suffering from stroke.
What we discovered was quite promising. The study suggested that these retinoic acid-loaded nanoparticles have the potential to promote protective measures for the brain's vascular system during a stroke. This could lead to significant advancements in stroke treatments, emphasizing the importance of vitamin A in neuroprotection.
Although these findings are encouraging, it's important to note that they were observed without direct comparison to other standard treatments. This means that while vitamin A showcases potential benefits, further research is needed to fully evaluate its effectiveness, particularly in combination with existing therapies.
What we discovered was quite promising. The study suggested that these retinoic acid-loaded nanoparticles have the potential to promote protective measures for the brain's vascular system during a stroke. This could lead to significant advancements in stroke treatments, emphasizing the importance of vitamin A in neuroprotection.
Although these findings are encouraging, it's important to note that they were observed without direct comparison to other standard treatments. This means that while vitamin A showcases potential benefits, further research is needed to fully evaluate its effectiveness, particularly in combination with existing therapies.
Read More
9
Study on vitamin A's stroke benefits
Retinoic acid exerts neuroprotective effects against focal cerebral ischemia by preventing apoptotic cell death.
High relevance to stroke treatment
We explored how retinoic acid, a vitamin A metabolite, impacts outcomes after stroke. The study involved adult male rats that underwent a procedure to simulate focal cerebral ischemia, which is essentially a model for stroke. We used a method that restricted blood flow to the brain to create a condition similar to what happens during a stroke, and then we examined the effects of retinoic acid compared to a control group.
After treating the rats with retinoic acid prior to the stroke simulation, we observed several positive outcomes. The treatment significantly reduced neurological deficits, brain swelling, and the volume of brain tissue affected by the stroke. Additionally, we found that retinoic acid decreased cell death in the brain, which is crucial in stroke recovery.
Notably, retinoic acid helped manage the balance between certain proteins that regulate cell survival and death. By increasing protective proteins and decreasing those that promote apoptosis (or cell death), we saw a restoration in the healthy balance disrupted by stroke. This exciting evidence suggests the potential of retinoic acid as a therapeutic option in stroke treatment through its action on apoptosis pathways.
After treating the rats with retinoic acid prior to the stroke simulation, we observed several positive outcomes. The treatment significantly reduced neurological deficits, brain swelling, and the volume of brain tissue affected by the stroke. Additionally, we found that retinoic acid decreased cell death in the brain, which is crucial in stroke recovery.
Notably, retinoic acid helped manage the balance between certain proteins that regulate cell survival and death. By increasing protective proteins and decreasing those that promote apoptosis (or cell death), we saw a restoration in the healthy balance disrupted by stroke. This exciting evidence suggests the potential of retinoic acid as a therapeutic option in stroke treatment through its action on apoptosis pathways.
Read More
9
Vitamin A and D improve stroke outcomes
Combination of vitamin A and D supplementation for ischemic stroke: effects on interleukin-1ß and clinical outcome.
Moderate relevance to vitamin A effects
We designed a study to explore how a combination of vitamin A and D affects stroke patients, particularly focusing on their serum levels of interleukin-1β, a marker of inflammation, and overall clinical outcomes. Over the course of 12 weeks, we monitored 120 ischemic stroke patients at Adam Malik Hospital, dividing them into four groups: those receiving only vitamin A, those receiving only vitamin D, those getting both vitamins, and a placebo group.
Our findings revealed that patients in the combination group exhibited significant increases in serum levels of both vitamins A and D compared to other groups. Importantly, we also noted a substantial drop in IL-1β levels, which is promising since this cytokine is linked to inflammation and the progression of atherosclerosis—an underlying factor in strokes.
The clinical outcomes were particularly encouraging, as the combination group showed the greatest improvement in stroke severity, as measured by the NIHSS, with the highest reduction in scores here. Overall, our study suggests that combining vitamin A with vitamin D could positively influence recovery and reduce inflammation in ischemic stroke patients.
Our findings revealed that patients in the combination group exhibited significant increases in serum levels of both vitamins A and D compared to other groups. Importantly, we also noted a substantial drop in IL-1β levels, which is promising since this cytokine is linked to inflammation and the progression of atherosclerosis—an underlying factor in strokes.
The clinical outcomes were particularly encouraging, as the combination group showed the greatest improvement in stroke severity, as measured by the NIHSS, with the highest reduction in scores here. Overall, our study suggests that combining vitamin A with vitamin D could positively influence recovery and reduce inflammation in ischemic stroke patients.
Read More
User Reviews
USERS' SCORE
Good
Based on 1 Review
8.5
- All Reviews
- Positive Reviews
- Negative Reviews
8.8
Improved health condition
Following my mother's stroke on her 62nd birthday and my father's at 58, I was diagnosed with vascular disease at 37. After struggling to change my diet, I began a homeopathic regimen of three pills daily. Six months later, I was able to stop my blood pressure medication, and my health has significantly improved, as has my skin. My doctor supports my lifestyle changes, and I now feel healthier internally.
Read More
