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SCIENTIFIC SCORE
Possibly Effective
Based on 52 Researches
7.6
USERS' SCORE
Good
Based on 3 Reviews
8.6
Supplement Facts
Serving Size: 1 Teaspoon (5 ml)
Amount Per Serving
%DV
Calories
40
Total Fat
4.5 g
6%**
Saturated Fat
0.5 g
3%**
Cholesterol
15 mg
5%
Vitamin A (from cod liver oil and retinyl palmitate)
255 mcg RAE(850 IU)
28%
Vitamin D (from cod liver oil and cholecalciferol)
10 mcg (400 IU)
50%
Vitamin E (as d-alpha tocopherol)
6.7 mg
45%
Norwegian Cod Liver Oil
4.6 g
†
Total Omega-3 Fatty Acids*
1,100 mg
†
DHA (Docosahexaenoic Acid)*
500 mg
†
EPA (Eicosapentaenoic Acid)*
370 mg
†
DPA (Docosapentaenoic Acid)*
50 mg
†
Top Medical Research Studies
9
Cod liver oil aids heart stability
[Correction of disorders of cardiac electric stability in post-infarction cardiosclerosis using a diet enriched with polyunsaturated fatty acids].
Study shows promising results
We examined how cod liver oil might affect heart health following a heart attack. In our study, we fed rats a special form of cod liver oil called "eikonole," which is enriched with important fatty acids. After a period of feeding, we induced a heart attack and then looked at how the hearts functioned over the next month.
Our findings were promising. We observed that eikonole raised the heart's fibrillation threshold by 50%. This means that it took a stronger stimulus to trigger heart disturbances, which is a good sign for heart stability. Additionally, there was a significant reduction in both spontaneous extra heartbeats and those triggered by stimulation of the vagus nerve.
However, while eikonole improved certain aspects of heart function, it did not significantly change the overall pressure or speed of heart contractions. This suggests that while there are benefits, they may be limited to specific areas of cardiac function rather than an overall enhancement of heart performance.
Our findings were promising. We observed that eikonole raised the heart's fibrillation threshold by 50%. This means that it took a stronger stimulus to trigger heart disturbances, which is a good sign for heart stability. Additionally, there was a significant reduction in both spontaneous extra heartbeats and those triggered by stimulation of the vagus nerve.
However, while eikonole improved certain aspects of heart function, it did not significantly change the overall pressure or speed of heart contractions. This suggests that while there are benefits, they may be limited to specific areas of cardiac function rather than an overall enhancement of heart performance.
Read More
9
DHA aids post-heart attack recovery
The polyunsaturated fatty acids, EPA and DHA, ameliorate myocardial infarction-induced heart failure by inhibiting p300-HAT activity in rats.
Relevant to myocardial infarction treatments
We explored the effects of docosahexaenoic acid (DHA) on heart attack recovery in rats. The study aimed to understand how DHA, alongside eicosapentaenoic acid (EPA), can influence heart failure following myocardial infarction (MI).
Using several groups of rats experiencing moderate heart issues, we evaluated how these omega-3 fatty acids impacted heart function. We found that both DHA and EPA effectively curtailed the hypertrophic response in heart cells. This response is a significant factor in heart failure, where heart tissue thickens and hardens.
Notably, both DHA and EPA inhibited the activity of a histone acetyltransferase called p300. This activity is linked to molecular changes that promote heart cell enlargement and fibrosis. In our analysis, we observed that these fatty acids not only preserved cardiac function but also prevented structural changes common after a heart attack.
Overall, we noted that DHA had a comparable protective effect to EPA, significantly improving heart health and reducing fibrosis in the heart tissue. As such, the findings suggest that incorporating DHA could be a heart-friendly choice post-heart attack.
Using several groups of rats experiencing moderate heart issues, we evaluated how these omega-3 fatty acids impacted heart function. We found that both DHA and EPA effectively curtailed the hypertrophic response in heart cells. This response is a significant factor in heart failure, where heart tissue thickens and hardens.
Notably, both DHA and EPA inhibited the activity of a histone acetyltransferase called p300. This activity is linked to molecular changes that promote heart cell enlargement and fibrosis. In our analysis, we observed that these fatty acids not only preserved cardiac function but also prevented structural changes common after a heart attack.
Overall, we noted that DHA had a comparable protective effect to EPA, significantly improving heart health and reducing fibrosis in the heart tissue. As such, the findings suggest that incorporating DHA could be a heart-friendly choice post-heart attack.
Read More
9
Eicosapentaenoic acid aids heart recovery
Eicosapentaenoic Acid Preserves Mitochondrial Quality and Attenuates Cardiac Remodeling After Myocardial Infarction in Rats.
Significant effects on cardiac remodeling
We investigated how eicosapentaenoic acid (EPA) may help improve heart health after a heart attack. In our study, we induced myocardial infarction (MI) in male rats by ligating their coronary artery. Some of these rats received daily treatment with EPA, while others did not, allowing us to compare the effects.
Over 12 weeks, we observed that the rats treated with EPA showed better left ventricular function—essentially, their hearts were working more efficiently. Moreover, these rats had higher levels of EPA in their mitochondria, which are the energy factories of cells. Despite the damage caused by the heart attack, the EPA treatment helped maintain crucial energy levels and kept the mitochondrial function from declining, preserving a specific protein linked to cellular health.
Our findings suggest that including EPA in the diet can bolster mitochondrial quality and support heart function after an MI. This could mean that EPA may serve as an important dietary addition for promoting heart health following heart events.
Over 12 weeks, we observed that the rats treated with EPA showed better left ventricular function—essentially, their hearts were working more efficiently. Moreover, these rats had higher levels of EPA in their mitochondria, which are the energy factories of cells. Despite the damage caused by the heart attack, the EPA treatment helped maintain crucial energy levels and kept the mitochondrial function from declining, preserving a specific protein linked to cellular health.
Our findings suggest that including EPA in the diet can bolster mitochondrial quality and support heart function after an MI. This could mean that EPA may serve as an important dietary addition for promoting heart health following heart events.
Read More
Most Useful Reviews
9.5
Enhanced clarity
After taking this, I notice my thoughts are clearer and I have better memory retention. Norwegian snow cod liver oil is highly beneficial for lowering cholesterol and protecting against heart attack. I strongly recommend it for its numerous benefits.
Read More
9
Lowered cholesterol
Fish oil is excellent! It's far superior to pharmacy products. I buy it for both myself and my child; it helps reduce my cholesterol and improves my heart function, while it strengthens my child's immunity. The delivery is prompt and well-packaged.
Read More
8.8
Reduced inflammation
Omg! This oil is fantastic! I previously took cod liver oil in capsules, but after trying this oil, I prefer it. I feel a noticeable difference in my heart and brain health within a week, and my toe inflammation has improved significantly. I will definitely buy it again.
Read More
Medical Researches
SCIENTIFIC SCORE
Possibly Effective
Based on 52 Researches
7.6
9
DHA promotes heart recovery post-MI
Docosahexaenoic Acid-Enhanced Autophagic Flux Improves Cardiac Dysfunction after Myocardial Infarction by Targeting the AMPK/mTOR Signaling Pathway.
Moderate relevance to cod liver oil
We investigated how docosahexaenoic acid (DHA), a key component found in cod liver oil, might help in recovery after a heart attack. Our study utilized models of heart injury, including cultured heart cells and living mice, to observe the influence of DHA on heart health.
We found that DHA significantly enhanced the survival of heart cells exposed to low oxygen levels, a common condition after a heart attack. Additionally, in mice suffering from heart attacks, DHA reduced the heart’s damaged area and improved overall heart function while lessening scarring in the heart tissue.
One of the exciting findings was how DHA promoted autophagy—the body's way of cleaning out damaged cells—by activating a specific signaling pathway known as AMPK/mTOR. Blocking this autophagy process with an inhibitor diminished DHA's protective benefits, indicating that enhancing autophagy is a crucial way DHA helps the heart recover.
Our findings highlight that DHA can play an invaluable role in addressing heart damage following a heart attack, potentially offering a new avenue for treatment.
We found that DHA significantly enhanced the survival of heart cells exposed to low oxygen levels, a common condition after a heart attack. Additionally, in mice suffering from heart attacks, DHA reduced the heart’s damaged area and improved overall heart function while lessening scarring in the heart tissue.
One of the exciting findings was how DHA promoted autophagy—the body's way of cleaning out damaged cells—by activating a specific signaling pathway known as AMPK/mTOR. Blocking this autophagy process with an inhibitor diminished DHA's protective benefits, indicating that enhancing autophagy is a crucial way DHA helps the heart recover.
Our findings highlight that DHA can play an invaluable role in addressing heart damage following a heart attack, potentially offering a new avenue for treatment.
Read More
9
Cod liver oil aids heart stability
[Correction of disorders of cardiac electric stability in post-infarction cardiosclerosis using a diet enriched with polyunsaturated fatty acids].
Study shows promising results
We examined how cod liver oil might affect heart health following a heart attack. In our study, we fed rats a special form of cod liver oil called "eikonole," which is enriched with important fatty acids. After a period of feeding, we induced a heart attack and then looked at how the hearts functioned over the next month.
Our findings were promising. We observed that eikonole raised the heart's fibrillation threshold by 50%. This means that it took a stronger stimulus to trigger heart disturbances, which is a good sign for heart stability. Additionally, there was a significant reduction in both spontaneous extra heartbeats and those triggered by stimulation of the vagus nerve.
However, while eikonole improved certain aspects of heart function, it did not significantly change the overall pressure or speed of heart contractions. This suggests that while there are benefits, they may be limited to specific areas of cardiac function rather than an overall enhancement of heart performance.
Our findings were promising. We observed that eikonole raised the heart's fibrillation threshold by 50%. This means that it took a stronger stimulus to trigger heart disturbances, which is a good sign for heart stability. Additionally, there was a significant reduction in both spontaneous extra heartbeats and those triggered by stimulation of the vagus nerve.
However, while eikonole improved certain aspects of heart function, it did not significantly change the overall pressure or speed of heart contractions. This suggests that while there are benefits, they may be limited to specific areas of cardiac function rather than an overall enhancement of heart performance.
Read More
9
DHA aids post-heart attack recovery
The polyunsaturated fatty acids, EPA and DHA, ameliorate myocardial infarction-induced heart failure by inhibiting p300-HAT activity in rats.
Relevant to myocardial infarction treatments
We explored the effects of docosahexaenoic acid (DHA) on heart attack recovery in rats. The study aimed to understand how DHA, alongside eicosapentaenoic acid (EPA), can influence heart failure following myocardial infarction (MI).
Using several groups of rats experiencing moderate heart issues, we evaluated how these omega-3 fatty acids impacted heart function. We found that both DHA and EPA effectively curtailed the hypertrophic response in heart cells. This response is a significant factor in heart failure, where heart tissue thickens and hardens.
Notably, both DHA and EPA inhibited the activity of a histone acetyltransferase called p300. This activity is linked to molecular changes that promote heart cell enlargement and fibrosis. In our analysis, we observed that these fatty acids not only preserved cardiac function but also prevented structural changes common after a heart attack.
Overall, we noted that DHA had a comparable protective effect to EPA, significantly improving heart health and reducing fibrosis in the heart tissue. As such, the findings suggest that incorporating DHA could be a heart-friendly choice post-heart attack.
Using several groups of rats experiencing moderate heart issues, we evaluated how these omega-3 fatty acids impacted heart function. We found that both DHA and EPA effectively curtailed the hypertrophic response in heart cells. This response is a significant factor in heart failure, where heart tissue thickens and hardens.
Notably, both DHA and EPA inhibited the activity of a histone acetyltransferase called p300. This activity is linked to molecular changes that promote heart cell enlargement and fibrosis. In our analysis, we observed that these fatty acids not only preserved cardiac function but also prevented structural changes common after a heart attack.
Overall, we noted that DHA had a comparable protective effect to EPA, significantly improving heart health and reducing fibrosis in the heart tissue. As such, the findings suggest that incorporating DHA could be a heart-friendly choice post-heart attack.
Read More
9
DHA supports heart recovery post-MI
Metabolic transformation of fat in obesity determines the inflammation resolving capacity of splenocardiac and cardiorenal networks in heart failure.
Study shows DHA's positive role
We investigated how docosahexaenoic acid (DHA), a type of omega-3 fatty acid, influences recovery following a heart attack (MI) in the context of dietary fat intake. The study began by feeding mice with safflower oil (rich in omega-6 fatty acids) for 12 weeks, followed by DHA supplementation for another 8 weeks before inducing a heart attack.
Through this process, we observed that the early intake of safflower oil led to heightened inflammation, delaying the body’s ability to heal. However, when we supplemented with DHA, we noted a favorable shift. DHA increased the levels of specialized proresolving mediators (SPMs) that help the body resolve inflammation. These mediators seemed to counteract the negative effects of safflower oil by enhancing mechanisms in both the heart and kidneys critical for recovery post-MI.
Additionally, DHA contributed to an increase in resolving macrophages, which play a vital role in repairing the heart, and it also elevated T regulatory cells in the heart tissue during chronic heart failure. This might suggest that transitioning from a diet high in omega-6 fatty acids to one rich in omega-3s like DHA could improve outcomes after heart attacks.
Overall, while excessive safflower oil intake worsens inflammation and affects heart recovery, DHA promotes a healthier resolving phase, supporting better heart and kidney function in the aftermath of a heart attack.
Through this process, we observed that the early intake of safflower oil led to heightened inflammation, delaying the body’s ability to heal. However, when we supplemented with DHA, we noted a favorable shift. DHA increased the levels of specialized proresolving mediators (SPMs) that help the body resolve inflammation. These mediators seemed to counteract the negative effects of safflower oil by enhancing mechanisms in both the heart and kidneys critical for recovery post-MI.
Additionally, DHA contributed to an increase in resolving macrophages, which play a vital role in repairing the heart, and it also elevated T regulatory cells in the heart tissue during chronic heart failure. This might suggest that transitioning from a diet high in omega-6 fatty acids to one rich in omega-3s like DHA could improve outcomes after heart attacks.
Overall, while excessive safflower oil intake worsens inflammation and affects heart recovery, DHA promotes a healthier resolving phase, supporting better heart and kidney function in the aftermath of a heart attack.
Read More
9
DHA supports heart recovery
Docosahexaenoic Acid-Enhanced Autophagic Flux Improves Cardiac Dysfunction after Myocardial Infarction by Targeting the AMPK/mTOR Signaling Pathway.
Strong focus on DHA effects
We set out to explore how docosahexaenoic acid (DHA), a beneficial omega-3 fatty acid found in fish oil and seaweed, might play a role in heart health, particularly after a heart attack. Our findings stemmed from experiments on both isolated heart cells under low oxygen conditions and live mice experiencing myocardial infarction (MI), which is the medical term for a heart attack.
The results were promising. We observed that DHA not only improved cell survival in stressed heart cells but also minimized damage in mice following a heart attack. Specifically, we noted a reduction in heart injury and a boost in heart function, highlighting DHA's potential as a protective agent.
Importantly, we found that DHA enhances autophagy—a natural process that cleans out damaged cells—by activating specific signaling pathways in the body. We established that in both our test models, the presence of DHA led to less cell death and richer heart function recovery. However, when we inhibited the autophagy process in experiments, the protective advantages of DHA were lost, underscoring its reliance on this cell-cleaning mechanism.
Our study indicates that DHA may serve as a valuable aid in healing the heart after a heart attack by promoting processes that protect against cell damage. Balancing scientific insight with real-world implications offers a promising avenue for heart health strategies, especially for those recovering from myocardial infarction.
The results were promising. We observed that DHA not only improved cell survival in stressed heart cells but also minimized damage in mice following a heart attack. Specifically, we noted a reduction in heart injury and a boost in heart function, highlighting DHA's potential as a protective agent.
Importantly, we found that DHA enhances autophagy—a natural process that cleans out damaged cells—by activating specific signaling pathways in the body. We established that in both our test models, the presence of DHA led to less cell death and richer heart function recovery. However, when we inhibited the autophagy process in experiments, the protective advantages of DHA were lost, underscoring its reliance on this cell-cleaning mechanism.
Our study indicates that DHA may serve as a valuable aid in healing the heart after a heart attack by promoting processes that protect against cell damage. Balancing scientific insight with real-world implications offers a promising avenue for heart health strategies, especially for those recovering from myocardial infarction.
Read More
User Reviews
USERS' SCORE
Good
Based on 3 Reviews
8.6
9.5
Enhanced clarity
After taking this, I notice my thoughts are clearer and I have better memory retention. Norwegian snow cod liver oil is highly beneficial for lowering cholesterol and protecting against heart attack. I strongly recommend it for its numerous benefits.
9
Lowered cholesterol
Fish oil is excellent! It's far superior to pharmacy products. I buy it for both myself and my child; it helps reduce my cholesterol and improves my heart function, while it strengthens my child's immunity. The delivery is prompt and well-packaged.
8.8
Reduced inflammation
Omg! This oil is fantastic! I previously took cod liver oil in capsules, but after trying this oil, I prefer it. I feel a noticeable difference in my heart and brain health within a week, and my toe inflammation has improved significantly. I will definitely buy it again.
